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- Highly Upregulated Lhx2 in the Foxn1−/− Nude Mouse Phenotype Reflects a Dysregulated and Expanded Epidermal Stem Cell Niche
- Hair follicle aging is driven by transepidermal elimination of stem cells via COL17A1 proteolysis
- Phospholipase C‐δ1 is an essential molecule downstream of <i>Foxnl,</i> the gene responsible for the nude mutation, in normal hair development
- Mesenchymal Meis2 controls whisker development independently from trigeminal sensory innervation
- Perspectives of Alopecia behind the Regulation of Foxn1 Gene Exposes the Human Nude Phenotype
- Control of hair follicle cell fate by underlying mesenchyme through a CSL–Wnt5a–FoxN1 regulatory axis
- Hairless-knockout piglets generated using the clustered regularly interspaced short palindromic repeat/CRISPR-associated-9 exhibit abnormalities in the skin and thymus
- FOXO1, TGF-β Regulation and Wound Healing
- Morphogenesis and maintenance of the 3D thymic medulla and prevention of nude skin phenotype require FoxN1 in pre- and post-natal K14 epithelium
- Molecular biology of hair morphogenesis: Development and cycling
- Learning from nudity: lessons from the nude phenotype
- FOXN1: A Master Regulator Gene of Thymic Epithelial Development Program
- The Nude Mutant Gene Foxn1 Is a HOXC13 Regulatory Target during Hair Follicle and Nail Differentiation
- Genetic interplays between Msx2 and Foxn1 are required for Notch1 expression and hair shaft differentiation
- Foxn1 in Skin Development, Homeostasis and Wound Healing
- Biological significance of FoxN1 gain-of-function mutations during T and B lymphopoiesis in juvenile mice
- Secretory phospholipase A2-IIA overexpressing mice exhibit cyclic alopecia mediated through aberrant hair shaft differentiation and impaired wound healing response
- Foxp1 Regulates the Proliferation of Hair Follicle Stem Cells in Response to Oxidative Stress during Hair Cycling
- The role of androgen and androgen receptor in skin-related disorders
- Keratin K15 as a Biomarker of Epidermal Stem Cells
- Two Mechanisms Regulate Keratin K15 Expression In Keratinocytes: Role of PKC/AP-1 and FOXM1 Mediated Signalling
- P63 targeted deletion under the FOXN1 promoter disrupts pre-and post-natal thymus development, function and maintenance as well as induces severe hair loss
- Identification of an Intronic Regulatory Element Necessary for Tissue-Specific Expression of <i>Foxn1</i> in Thymic Epithelial Cells
- Gsdma3 is required for hair follicle differentiation in mice
- The nude gene and the skin
- Desmoglein 4 is regulated by transcription factors implicated in hair shaft differentiation
- Gata6 promotes hair follicle progenitor cell renewal by genome maintenance during proliferation
- Transcriptional regulation of the thymus master regulator <i>Foxn1</i>
- VDR is an essential regulator of hair follicle regression through the progression of cell death
- Deletion of the epidermis derived laminin γ1 chain leads to defects in the regulation of late hair morphogenesis