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Fibromodulin treatment helps reduce scarring and improves wound healing by making it more like fetal healing.

The African spiny mouse heals skin without scarring due to different protein activity compared to the common house mouse, which heals with scarring.

Effective PCOS treatments require targeting specific signaling pathways.

Cells from the lower part of hair follicles are a promising, less invasive option for immune system therapies.

DPP4-positive fibroblasts play a major role in producing proteins that lead to skin fibrosis.

Different types of fibroblasts play various roles in both healthy and diseased tissues, and understanding them better could improve treatments for fibrotic diseases.

Scarless healing is complex and influenced by genetics and environment, while better understanding could improve scar treatment.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

PBMCsec can help reduce and improve thick skin scars.

Radiation treatment causes skin fibrosis by increasing certain fibroblast subpopulations, but using a c-Jun inhibitor or fat grafting can reduce this effect.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.

Antiviral drugs, especially daclatasvir, may be a new treatment for a rare skin disease, improving survival and reducing symptoms in mice.

Fat cells help recruit healing cells and build skin structure during wound healing.

TRPV4 helps cells repair tissue and reduce scarring by controlling calcium levels.

Printing human stem cells and a special matrix during surgery can help grow new skin and hair-like structures in rats.

The conclusion is that accurately replicating the complexity of the extracellular matrix in the lab is crucial for creating realistic human tissue models.

Changing the environment around stem cells could help tissue repair, but it's hard to be precise and avoid side effects.

Bacteria can help skin regenerate through a process called IL-1β signaling.

Calreticulin has roles in healing, immune response, and disease beyond its known functions in the endoplasmic reticulum.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Fat-derived stem cells show promise for skin repair and reducing aging signs but need more research for consistent results.

Fetal skin heals without scarring due to unique cells and processes not present in adult skin healing.

Mammals might fail to regenerate not because they lack the right cells, but because of how cells respond to their surroundings, and changing this environment could enhance regeneration.

Different types of facial fat affect aging and treatment outcomes; more research is needed to enhance anti-aging procedures.

Wound healing insights can improve regenerative medicine.

Hydrogels could lead to better treatments for wound healing without scars.

Researchers developed a 3D model of human hair follicle cells that can help understand hair growth and test new hair loss treatments.

Fetal wounds heal without scarring because of different biological factors, which could help improve adult wound healing.

Wounds heal without scarring in early development but later result in scars, and studying Wnt signaling could help control scarring.

Lanyu pigs show that partial-thickness wounds can partially regenerate important skin structures, which may help improve human skin healing.