Search

everythinglearnresearchcommunity

for

Search:↓

The African spiny mouse heals skin without scarring due to different protein activity compared to the common house mouse, which heals with scarring.

Fibromodulin treatment helps reduce scarring and improves wound healing by making it more like fetal healing.

Effective PCOS treatments require targeting specific signaling pathways.

Cells from the lower part of hair follicles are a promising, less invasive option for immune system therapies.

TRPV4 helps cells repair tissue and reduce scarring by controlling calcium levels.

Different types of fibroblasts play various roles in both healthy and diseased tissues, and understanding them better could improve treatments for fibrotic diseases.

Scarless healing is complex and influenced by genetics and environment, while better understanding could improve scar treatment.

DPP4-positive fibroblasts play a major role in producing proteins that lead to skin fibrosis.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

PBMCsec can help reduce and improve thick skin scars.

Radiation treatment causes skin fibrosis by increasing certain fibroblast subpopulations, but using a c-Jun inhibitor or fat grafting can reduce this effect.

Certain genes are more active during wound healing in axolotl and Acomys, which could help develop materials that improve human wound healing and regeneration.

Bacteria can help skin regenerate through a process called IL-1β signaling.

Antiviral drugs, especially daclatasvir, may be a new treatment for a rare skin disease, improving survival and reducing symptoms in mice.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.

The dataset includes detailed genetic information from mouse skin cells before and after injury.

The dataset includes detailed genetic information from mouse skin cells before and after injury.

Disrupting YAP signaling in skin cells leads to scar-free healing directed by specific cell signals.

Changing the environment around stem cells could help tissue repair, but it's hard to be precise and avoid side effects.

MSCs and their exosomes may speed up skin wound healing but need more research for consistent use.

Verteporfin treatment in mice led to complete skin healing without scarring.

Fat cells help recruit healing cells and build skin structure during wound healing.

Immune cells are essential for skin regeneration using biomaterial scaffolds.

Understanding how baby skin heals without scars could help develop treatments for adults to heal wounds without leaving scars.

Fat-derived stem cells show promise for skin repair and reducing aging signs but need more research for consistent results.

Calreticulin has roles in healing, immune response, and disease beyond its known functions in the endoplasmic reticulum.

The conclusion is that accurately replicating the complexity of the extracellular matrix in the lab is crucial for creating realistic human tissue models.

Hyaluronic acid and versican are important for skin healing and hair growth and might help in regenerative medicine.

Using developmental signaling pathways could improve adult wound healing by mimicking scarless embryonic healing.

Printing human stem cells and a special matrix during surgery can help grow new skin and hair-like structures in rats.