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New compounds were found to help increase hair growth and decrease hair loss.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Eating high-glycemic foods and drinking milk may worsen acne by increasing insulin and IGF-1 levels.

miR-31 regulates hair growth by controlling gene expression in hair follicles.

Future treatments for androgenic alopecia may focus on reactivating hair follicle stem cells and improving drug delivery.

Disabling the FGF5 gene in sheep leads to longer wool.

FGF5 gene mutations cause unusually long eyelashes by affecting hair growth regulation.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Estrogens significantly influence hair growth by interacting with receptors in hair follicles and may help regulate the hair growth cycle.

Growth factors and cytokines are important for hair growth and could potentially treat hair loss, but more research is needed to overcome challenges before they can be used in treatments.

The TGF-β family helps control how cells change and move, affecting skin, hair, and organ development.

Targeted cancer therapies have a significant but lower risk of causing hair loss compared to chemotherapy.

Found 32 genes linked to male baldness, affecting hair growth and stress-related pathways.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.

Hair, teeth, and mammary glands develop similarly at first but use different genes later.

Mutant mice help researchers understand hair growth and related genetic factors.

Scalp cooling is the most effective FDA-approved method to prevent chemotherapy-induced hair loss, but more research is needed for other treatments.

PI3K/Akt pathway is crucial for hair growth and regeneration.

Activin activation in skin cells speeds up wound healing without affecting scar quality.

The skin and thymus develop similarly to protect and support immunity.

Some growth factors, while important for normal body functions, can cause diseases when not regulated properly.

Certain genes are more active in baby scalp cells and can help grow hair when added to adult mouse skin cells.

Overexpression of activin A in mice skin causes skin thickening, fibrosis, and improved wound healing.

TGF-β signaling is crucial for stem cell maintenance, differentiation, and has implications for cancer treatment.

Chemotherapy often causes temporary hair loss, which is distressing and needs better treatment and support.

Hair usually grows back 1-3 months after treatment for anagen effluvium, and children with Loose Anagen Hair Syndrome often improve by adolescence.

Cyclosporin A helps mice grow hair by blocking a specific protein activity in skin cells.

MicroRNAs are crucial for controlling skin development and healing by regulating genes.

FLRG and follistatin have different roles in wound healing.

White hair grows thicker and faster than black hair due to higher activity of growth-related genes and proteins.