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The study found that tight junctions reach the top layer of the skin's stratum granulosum, not just the second top layer as previously thought.

Mouse skin fibroblasts vary in function and adaptability based on their environment.

Blocking EGFR in skin cells doesn't majorly increase inflammation markers.

TCDD speeds up skin barrier formation by increasing certain gene expressions.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

Activating Notch in adult skin causes T cells and neural crest cells to gather, leading to skin issues.

ACBP is essential for healthy skin and fur by maintaining the skin's barrier function.

ILK is essential for skin development, pigmentation, and healing.

Skin stem cells remember past inflammation, helping them respond better to future injuries and possibly aiding in treating skin issues.

Skin may help in getting rid of excess iron through the process of skin cell renewal.

A new treatment using AGED to modulate PPAR-γ shows promise for treating scarring hair loss by protecting and repairing hair follicle cells.

Many survivors of Stevens-Johnson Syndrome and Toxic Epidermal Necrolysis in Togo suffer long-term eye and skin problems.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.

Deleting MED1 in skin cells causes hair loss and skin changes.

N-acetyl-GED may help prevent and partially reverse a process that leads to scarring hair loss.

Epidermal Wnt/β-catenin signaling controls fat cell formation and hair growth.

Scientists discovered that certain stem cells from mice and humans can be used to grow new hair follicles and skin glands when treated with a special mixture.

ACBP is crucial for healthy skin in mice.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

Eating a high-fat fish oil diet caused mice to lose hair due to a specific immune cell activity in the skin linked to a protein called E-FABP.

PPARγ is crucial for skin health but can have both beneficial and harmful effects.

PPAR-γ helps control skin oil glands and inflammation, and its disruption can cause hair loss diseases.

Iris germanica rhizome-derived exosomes help protect skin cells from oxidative stress and aging.

Fat cells help recruit healing cells and build skin structure during wound healing.

The document concludes that skin stem cells are important for hair growth and wound healing, and could be used in regenerative medicine.

Epigenetic changes control how adult stem cells work and can lead to diseases like cancer if they go wrong.

Different types of stem cells in hair follicles play unique roles in wound healing and hair growth, with some stem cells not originating from existing hair follicles but from non-hair follicle cells. WNT signaling and the Lhx2 factor are key in creating new hair follicles.

Skin aging is due to impaired stem cell mobilization or fewer responsive stem cells.

Skin and hair can regenerate after injury due to changes in gene activity, with potential links to how cancer spreads. Future research should focus on how new hair follicles form and the processes that trigger their creation.