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High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.

Mechanical forces are crucial for shaping cells and forming tissues during development.

Nestin identifies specific progenitor cells in hair follicles that can become outer root sheath cells.

Hair growth environment recreated with challenges; stem cells make successful skin organoids.

Wounds can regenerate hair in young mice, but this ability declines with age, offering insights for improving tissue regeneration in the elderly.

Researchers found 617 genes that behave differently in cashmere goat hair follicles, which could help understand hair growth.

A20 protein is crucial for normal skin and hair development.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

Skin stem cells are crucial for maintaining and repairing the skin and hair, using a complex mix of signals to do so.

Epidermal stem cells are diverse and vary in activity, playing key roles in skin maintenance and repair.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

The PP2A-B55α protein is essential for brain and skin development in embryos.

Dermal EZH2 controls skin cell development and hair growth in mice.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

Hair follicles in the back of the rosette fancy mouse have reversed orientations due to a gene mutation.

Sweat gland development involves two unique skin cell programs and a temporary skin environment.

The exact identity of skin stem cells and how skin cells differentiate is not fully known.

Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

Dermal EZH2 controls skin cell growth and differentiation in mice.

Skin health and repair depend on the signals between skin stem cells and their surrounding cells.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

Blocking a specific protein signal can make hair grow on mouse nipples.

Too much Smad7 changes skin and hair development by breaking down a protein called β-catenin, leading to more oil glands and fewer hair follicles.

Foxi3 expression in developing teeth and hair is controlled by the ectodysplasin pathway.

Mice lacking a key DNA methylation enzyme in skin cells have a lower chance of activating stem cells necessary for hair growth, leading to progressive hair loss.

Scientists created complete hair-like structures by growing mouse skin cells together in a special gel.

A form of vitamin E promotes hair growth by activating a specific skin pathway.

Certain skin proteins can form anchoring structures without the protein AMACO.