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The document concludes that PCOS is a complex disorder caused by both genetic and environmental factors, affecting women's health in various ways, and requires personalized treatment.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

Exposure to too much androgen before birth might cause polycystic ovary syndrome later in life.

Insulin resistance is a key factor in polycystic ovary syndrome, but genetics may also contribute.

Obesity increases the risk of developing polycystic ovary syndrome, and weight loss can improve the condition.

A girl with rickets and hair loss was found to have a rare condition where her body didn't respond to vitamin D treatment.

The current methods for diagnosing polycystic ovary syndrome are too vague and may lead to misdiagnosis and problems in research.

Lifestyle changes and weight loss are key for treating PCOS-related metabolic issues and infertility, with various medications available for specific symptoms.

Different types of PCOS have different levels of metabolic problems, with the most severe type showing the highest disturbances.

Finasteride effectively reduces hair growth in women with polycystic ovary syndrome or idiopathic hirsutism.

Metformin should be used for PCOS mainly in those with glucose intolerance, and has limited benefits for infertility or hirsutism.

Woodhouse-Sakati syndrome often causes sexual development issues, hair loss, learning disabilities, deafness, muscle contractions, limb pain, and diabetes.

Young women with high male hormone levels or PCOS often feel more psychological distress and have a lower quality of life.

Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

An mFG score of 7 or higher indicates hirsutism in Filipino women, often linked to higher free testosterone levels.

Transgender patients need proper skin care, especially when undergoing hormone treatments, to manage issues like acne and hair loss.

The conclusion is that removing both ovaries is the best treatment for excess male hormones in postmenopausal women, with medication as another option, and managing insulin resistance is important for diagnosis and treatment.

Women with PCOS have higher 5α-reductase activity, which may be linked to insulin resistance.

Certain changes to the B-ring of androgen receptor ligands can increase their effectiveness for potential treatments of muscle and bone conditions.

Women with PCOS in Sicily show different levels of metabolic problems depending on their PCOS type, with obesity and abnormal lipid profiles being common.

The document concludes that PCOS has a strong genetic component, but more research is needed to fully understand the specific genes involved.

Older obese women with PCOS have higher cardiovascular and metabolic risks despite lower androgen levels.

The study suggests certain ACE gene variations are more common in women with PCOS and may be linked to increased insulin resistance.

A new gene, mrp4, is found in mice and may play a unique role in hair follicle development in tails and ears.

Obesity changes androgen levels in women with PCOS, leading to higher testosterone relative to androstenedione.

High levels of male hormones in women without tumors are linked to metabolic problems and can be treated with metformin.

Higher Lnk protein levels in ovaries may lead to insulin resistance in women with PCOS.

A woman with a rare adrenal tumor and hormonal disorder had improved testosterone levels after surgery, but her menstrual irregularities continued.

Young women with PCOS and no other heart risk factors have normal heart function.

The document concludes that treating androgen excess needs patience, managing expectations is important, and many drugs used are not officially approved, suggesting cosmetic options for mild cases.