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Targeting the protein Caveolin-1 might help treat a type of scarring hair loss called Frontal Fibrosing Alopecia.

Genetic mutations cause various hair diseases, and whole genome sequencing may reveal more about these conditions.

Any drug can cause skin reactions, but antibiotics, NSAIDs, and psychotropic drugs are more common, with some reactions being life-threatening.

Androgens increase growth factors in skin cells, which may lead to acne.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

The TRPV3 ion channel is important for skin and hair health and could be a target for treating skin conditions.

A new hydrogel with micronized amnion helps achieve better, scar-free skin healing.

Cancer treatment drugs can cause permanent hair loss by damaging hair follicle stem cells, but a specific inhibitor might reverse this effect.

EGFR deficiency causes significant changes in skin cells and hair follicles.

SOX4 is crucial for the development of melanoma.

Blocking EGFR can lead to hair loss due to inflammation and stem cell damage.

The study found that androgen receptors in skin cells mainly affect the focal adhesion pathway and control the caveolin-1 gene, with implications for new treatments for related diseases.

ILK and ELMO2 help cells move and stick together, important for wound healing and hair growth.

Baicalin may help treat hair loss by promoting hair follicle growth and activating specific cellular pathways.

Skin problems like acne, dry skin, and nail and hair changes are common in patients taking EGFR inhibitors.

STAT5 activation is crucial for starting the hair growth phase.

Skin side effects from EGFR inhibitor cancer treatment can be managed effectively, often without stopping the medication.

EGFR cancer drugs often cause a rash, which can lead to distress and isolation, but may also improve outcomes for some cancer patients. Current treatments for the rash are limited, but a compound called menadione shows promise.

Activating TRPV3 channels stops hair growth by killing hair follicle cells.

Dr. Mario Lacouture recommends preventive and specific treatments for skin side effects caused by EGFR inhibitors to improve patients' quality of life.

Activating TRPV3 stops human hair growth.

Y27632 increases cell growth through EGFR signaling, not ROCK1/2.

Activating TRPV3 reduces skin oil production and increases inflammation, potentially causing dry skin issues.

EGFR helps control how hair grows and forms without needing p53 protein.

Cutaneous Lymphadenoma is a unique skin tumor with specific protein markers and common gene mutations that may cause continuous cell growth.

Activating TLR9 helps heal wounds and regrow hair by using specific immune cells.

TRPV3 in skin cells causes inflammation and cell death.

Planarian regeneration begins with a specific gene activation caused by injury, essential for healing and tissue regrowth.

Coffee bean residue extract helps hair growth by activating cell processes.