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PRP injections can increase hair density in people with androgenic alopecia.

Abdominal skin heals faster than dorsal skin because it has more stem cells.

Peptide hydrogels show promise for healing skin, bone, and nerves but need improvement in stability and compatibility.

The model better predicts how water-loving and fat-loving substances move through the skin by including tiny pores and hair follicle paths.

Photobiomodulation therapy helps manage cancer treatment side effects but needs more research for optimization.

Basonuclin 2 is vital for the development of facial bones, hair follicles, and male germ cells in adult mice, and its absence can lead to dwarfism and abnormal follicles.

New synthetic polymers help improve skin wound healing and can be enhanced by adding natural materials and medicines.

Stem cells and their exosomes show promise for repairing tissues and healing wounds when delivered effectively, but more research is needed on their tracking and optimal use.

Topical corticosteroids are the main treatment for Erosive Pustular Dermatosis, but recurrence is common after stopping treatment.

Combining PRP with Minoxidil improves hair density and thickness more than Minoxidil alone.

Injectable treatments can effectively and safely improve hair growth in adults with androgenetic alopecia.

Epidermal stem cells create and maintain skin structures like hair and nails through specific signaling pathways and vary by location and function.

Markers help differentiate between apocrine and eccrine sweat glands to identify sweat gland tumors.

Hair follicle stem cells can form both hair follicles and skin.

Common variants in the Trichohyalin gene are linked to straight hair in Europeans.

Innate lymphoid cells help control skin bacteria by regulating sebaceous glands.

Early development of hair, teeth, and glands involves specific signaling pathways and cellular interactions.

Wnt signaling controls whether hair follicle stem cells stay inactive or regenerate hair.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

Rac1 is essential for proper hair structure and color.

Hair and nail cells share similar proteins, indicating a common differentiation pathway.

Cholesterol sulfate buildup due to a genetic mutation disrupts the skin barrier, leading to the scaling skin seen in X-linked ichthyosis.

miR-29a-5p prevents the formation of early hair structures by targeting a gene important for hair growth and is regulated by a complex network involving lncRNA627.1.

The research showed that CRISPR/Cas9 can fix mutations causing a skin disease in stem cells, which then improved skin grafts in mice, but more work on safety and efficiency is needed.

People with pattern hair loss have higher polyamine levels in the top of their head compared to the back.

The Wnt signaling pathway is not a major factor in the development of keratoacanthoma, a type of skin tumor.

Glycogen metabolism is important for energy and processes in human hair follicles, and hair follicles may produce glucose from lactate.

Stabilizing HIF1A in hair follicles increases glycolysis, which may help reduce oxidative stress and support hair growth.

The research suggests that a specific skin gene can be controlled by signals within and between cells and is wrongly activated in certain skin diseases.