Search

everythinglearnresearchcommunity

for

Search:↓

The document concludes that more research is needed to better understand and treat primary cicatricial alopecias, and suggests a possible reclassification based on molecular pathways.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Alopecia areata is an autoimmune condition causing hair loss, linked to genetic factors and immune system issues, with no cure yet.

Oleoylethanolamide (OEA) safely boosts fat production in skin cells and may help treat dry skin and reduce inflammation.

Toll-like receptors are important for wound healing, but can slow it down in diabetic wounds.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

Different immune responses cause hair loss in scalp diseases, with unique patterns in scalp psoriasis possibly protecting against hair loss.

Acne patients' skin, both with and without lesions, shows a strong immune response and higher antimicrobial activity.

Oxidative stress plays a significant role in alopecia areata, and new treatments may include JAK inhibitors and antioxidants.

Topical JAK inhibitors can effectively treat alopecia areata and vitiligo by modulating immune responses.

Vitamin A affects hair loss and immune response in alopecia areata.

Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

CXCL12 protein slows down hair growth through its receptor CXCR4. Blocking this can potentially increase hair growth.

The document concludes that stem cells and their environments are crucial for skin and hair health and have potential for medical treatments.

The secretions of mesenchymal stem cells could be used for healing without using the cells themselves.

The study found that immune responses disrupt hair growth cycles, causing hair loss in alopecia areata.

Alopecia areata, a type of hair loss, is likely an autoimmune disease with a genetic link, but its exact cause is still unknown.

CD2 might be a new treatment target for patchy alopecia areata.

Some drugs, especially biologics, can cause skin reactions that look like other skin diseases, and stopping the drug usually helps clear up these reactions.

Faulty inflammasome activation may lead to autoimmune skin diseases and could be a target for new treatments.

Patients with RASopathies have a higher risk of autoimmune disorders and should be routinely screened.

Claudin-1 and Claudin-3 are crucial for keeping hair follicle structure and preventing a type of hair loss called telogen effluvium.

Mint water extract boosts protective enzymes in skin cells.

Nicotinic acid reduces excessive oil production in skin cells by activating a specific receptor, which could help treat acne.

The role of γδT-cells in causing alopecia areata remains unclear.

The document concludes that better targeted treatments are needed for wound healing, and single-cell technologies may improve cell-based therapies.

Lichen Planus is a less common condition affecting skin and mucous membranes, with various types and associated risk factors, challenging to diagnose, significantly impacts life quality, and may have a risk of cancerous changes in oral lesions.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.