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ILC1-like cells can cause alopecia areata by attacking hair follicles.

People with alopecia areata have more Th17 cells and fewer Treg cells, which may be key to the condition's development.

Special immune cells called Regulatory T cells help control skin inflammation and repair in various skin diseases.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

Androgens may influence T cells, contributing to higher autoimmune liver disease risk in women.

γδTregs can protect hair follicles from alopecia areata and may help regrow hair.

Certain immune cells contribute to skin autoimmune diseases, and some treatments can reverse hair loss in these conditions.

Using healthy donor stem cells can potentially calm overactive immune cells and reduce inflammation in severe hair loss patients, offering a possible treatment method.

Activating TLR9 helps heal wounds and regrow hair by using specific immune cells.

Women with PCOS have fewer activated T cells in their ovarian follicles, which might affect fertility.

Alopecia areata involves specific T-cells, unlike androgenetic alopecia.

Newborns with the common rash Erythema Toxicum have many active mast cells in their skin, but these cells don't produce the LL-37 peptide.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

Valproic Acid could potentially be used to treat immune-related conditions due to its ability to modify immune cell functions.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

Different immune cells like platelets, mast cells, neutrophils, macrophages, T cells, B cells, and innate lymphoid cells all play roles in skin wound healing, but more research is needed due to inconsistent results and the complex nature of the immune response.

Autoimmune hair loss may be linked to increased Th1 and Th17 cells and decreased Th2 cells.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

Vesiculobullous lesions should be considered part of canine cutaneous epitheliotropic T-cell lymphoma.

Increasing the levels of a protein called FGF9 can promote hair growth, but humans may not respond the same way due to a lack of certain cells.

Immune cells are crucial for hair growth and preventing hair loss.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Lower CD200R1 on certain immune cells is linked to more severe rheumatoid arthritis and immune imbalance.

ILC1 cells contribute to hair loss in alopecia areata.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.

UV light helped human hair transplants survive in mice without broad immunosuppression.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.