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A new semi-automatic hair implanter could make hair transplants easier, more successful, and more accessible.

Special particles from skin cells can promote hair growth by activating a specific growth signal.

Transplanting a mix of specific skin cells can significantly improve the repair of damaged hair follicles.

The research created a detailed map of skin cells, showing that certain cells in basal cell carcinoma may come from hair follicles and could help the cancer grow.

Hair growth can be induced by transplanting certain cells, but these cells lose their properties during culturing. The best cell interaction happens in a liquid medium under gravity, and using collagen doesn't help. Future research could focus on using growth factors to stimulate these cells.

The hair follicle is a great model for research to improve hair growth treatments.

Hair follicles have a more inactive cell cycle than other skin cells, which may help develop targeted therapies for skin diseases and cancer.

Certain miRNAs may play a role in sheep hair follicle development, which could help improve wool production.

The study found that a one-step antibody method is better than the LSAB method for accurately studying hair follicle structures without false positives.

Hair follicles can regenerate after radiation damage but not during a specific growth phase.

Three specific proteins can turn adult skin cells into hair-growing cells, suggesting a new hair loss treatment.

Cucurbitacin helps mice grow hair by blocking a protein that stops hair growth.

Stress stops hair growth in mice by causing early hair growth phase end and harmful inflammation through a specific nerve-related pathway.

The telogen phase of hair growth is active and important for preparing hair follicles for regeneration, not just a resting stage.

HSPGs help control stem cell behavior, affecting hair growth and offering a target for hair loss treatments.

The document concludes that more research is needed to better understand and treat primary cicatricial alopecias, and suggests a possible reclassification based on molecular pathways.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Micrografts improve hair density and thickness without side effects.

Minoxidil helps prevent stress-caused hair loss in mice.

Skin-derived stem cells grow faster and are easier to obtain than hair follicle stem cells, but both can become various cell types.

Blood-derived CD34+ cells speed up healing, reduce scarring, and regrow hair in skin wounds.

Cathepsin L is essential for normal hair growth and development.

Low fluence photoepilation temporarily removes hair by targeting the hair follicle's pigmented area without severe damage.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

The study found that the protein Dkk4 helps regulate hair growth by controlling Wnt signaling in mice.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

The study concluded that similar pathways regulate hair growth in dogs and mice, and these pathways are disrupted in dogs with Alopecia X, affecting stem cells and hormone metabolism.

Wound healing can help prevent hair loss from chemotherapy in young rats by increasing interleukin-1β signaling.

Higher light doses cause more damage to hair follicles, predicting better hair removal results.