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Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Human hair follicles can regenerate and recover after severe injury by going through a brief abnormal resting phase before growing again.

Grafted rodent and human cells can regenerate hair follicles, but efficiency decreases with age.

Older thin hair is not just thinner but also has different shape, structure, and stiffness.

Human nails and hair follicles have similar gene activity, especially in the cells that contribute to their growth and development.

Scientists successfully created mouse hair proteins in the lab, which are stable and similar to natural hair.

Microfibrils are key for permanent waves, and hydrolyzed keratin improves wave formation and hair condition.

KAP genes are crucial for hair development and show both shared and unique traits in humans, chimpanzees, and baboons.

Understanding how cells die in the skin is important for treating skin diseases and preventing hair loss.

Researchers created early-stage hair-like structures from skin cells, showing how these cells can self-organize, but more is needed for complete hair growth.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

α3β1-integrin is crucial for maintaining normal hair follicle shape and function but not needed for the development of the surrounding skin.

Autophagy helps keep skin healthy and may improve treatments for skin diseases.

POMC-derived peptides are important for skin functions like immune response and stress management.

Over 67 genes linked to ichthyosis help improve diagnosis and treatment.

Enterococcus faecalis delays wound healing by disrupting cell functions and creating an anti-inflammatory environment.

Foreskin keratinocytes and SVK14 cells express certain cytochrome P450 enzymes, unlike hair follicle keratinocytes.

The AVET system effectively delivers genes to human keratinocytes and may help treat skin diseases.

Different combinations of human hair keratins affect how hair fibers form.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Human stem cells can help form hair follicles in mice.

RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

A potential treatment for atopic dermatitis could be to increase PADI1 expression to improve skin barrier function.

The protein Par3 is crucial for healthy skin, affecting the skin barrier, cell differentiation, and stem cell maintenance.

Male hormones indirectly affect skin cell development by increasing growth factor levels from skin fibroblasts.

The study found that thioglycolic acid breaks down hair bonds more consistently than l-cysteine, which is less damaging to hair.

The KRTAP11-1 gene promoter is crucial for specific expression in sheep wool cortex.

Pathologic tissues have more soluble proteins than normal tissues.

M-CSF-stimulated myeloid cells can turn into skin cells and help heal wounds and regrow hair.