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The Rotterdam Study updated its design and objectives in 2012, providing insights into various diseases in the elderly, including skin cancer, bone health, liver disease, neurological and psychiatric conditions, and respiratory issues.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Certain gene variations in the Vitamin D Receptor are linked to higher risk of female hair loss.

Chinese domestic goats have unique genetic traits due to domestication and geographic isolation.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Melanoma's complexity requires personalized treatments due to key genetic mutations and tumor-initiating cells.

Higher miR-34a levels and the A variant of the MIR-34A gene are linked to increased risk and severity of alopecia areata.

Genes and hormones cause hair loss, with four genes contributing equally.

Hormones control reproduction and are crucial for body balance.

Parthenolide promotes hair growth in mice and may influence pathways related to male pattern baldness.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

CDP is crucial for lung and hair follicle cell development.

Male genital development is driven by androgen signaling and understanding it could help address congenital anomalies.

The research found genetic differences in identical twins that could explain why one twin has a disease while the other does not.

Androgen receptor signaling causes early aging of cells important for hair growth by damaging their DNA.

The conclusion is that skin and hair patterns are formed by a mix of cell activities, molecular signals, and environmental factors.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

Two young siblings experienced hair loss without hormone issues or other skin problems.

Molecular diagnostics help identify genetic defects causing endocrine diseases, improving diagnosis and treatment options.

The HoxC gene cluster and its enhancers are essential for developing hair and nails in mammals.

The Rotterdam Study investigates diseases in older adults and has produced many research findings.

Genetic variation in the androgen receptor gene mainly causes early-onset hair loss, with maternal inheritance playing a key role.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

Polycystic Ovary Syndrome likely starts in childhood and may be genetic and influenced by early hormone exposure.

Oestrogens help maintain healthy skin, heal wounds, and may protect against skin aging and cancer.

Reduced AR gene methylation may cause early pubic hair growth in girls.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

Testing for CAG repeat polymorphism in the androgen receptor gene is not currently recommended for managing hypogonadism.

Researchers identified specific genes that are important for mouse skin cell development and healing.