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Different sizes of keratin peptides can strengthen hair, with smaller ones possibly increasing volume and larger ones repairing damage.

TRα and CRABPII genes change their activity levels during goat fetal skin development.

Exosomes help hair follicle development in cashmere goats.

The absence of functional sebaceous glands causes hair follicle destruction and scarring alopecia.

KAP9.2 and Hoxc13 genes are important for cashmere growth and vary in activity during different stages.

Primary idiopathic pseudopelade of Brocq causes gradual, scarring hair loss with no effective treatment.

Hair color is determined by melanin produced and transferred in hair follicles.

Sostdc1 controls the size and number of hair and mammary gland structures.

Future hair cosmetics will be safer and more effective.

Reducing Tyrosinase prevents mature color pigment cells from forming in mouse hair.

The conclusion is that genetic testing is important for diagnosing and treating various genetic hair disorders.

Different fields of expertise must work together to better understand hair growth and create effective hair loss treatments.

Certain genes are linked to the quality of cashmere in goats.

Minoxidil and finasteride are effective for treating hair loss, with dutasteride showing potential but with side effects.

The document concludes that more research is needed to reduce frequent hospital visits, addiction medicine education improves with specific training, early breast cancer surgery findings are emerging, nipple smears are not very accurate, surgery for older melanoma patients doesn't extend life, a genetic condition in infants can often be treated with one drug, doctors are inconsistent with blood clot medication, a certain gene may protect against cell damage, muscle gene overexpression affects many other genes, and some mitochondrial genes are less active in mice with tumors.

The TRPV3 gene variant may cause the long-haired suri alpaca coat.

The research found genes and miRNAs that may control hair growth in Forest Musk Deer.

miR-29a-5p prevents the formation of early hair structures by targeting a gene important for hair growth and is regulated by a complex network involving lncRNA627.1.

Wool follicles are complex, involving interactions between different cell types and structures.

Researchers found genes for cysteine-rich proteins that form the protective layer of hair in humans and sheep.

Certain RNA molecules are important for the development of wool follicles in sheep.

Wool fibre curvature is due to longer orthocortical cells compared to paracortical cells.

Wool follicle cells are more complex than previously thought.

Wool follicles can grow in a lab with the right nutrients and conditions.

Merino sheep have fewer wool follicles at birth than before birth.

Wool follicles grew fibres for 8-10 days in a serum-free culture, influenced by calcium, glucose, amino acids, and insulin.

Wool and hair fibers absorb moisture similarly due to their keratin structure, with the amount of non-crystalline areas affecting the moisture uptake.

Sheep wool follicles absorb different amino acids at various rates and locations, which could affect wool growth based on diet and genetics.

Flumethasone causes wool shedding in Merino wethers, with recovery in about 60 days.

Sheep wool follicles can metabolize both glucose and acetate using different pathways important for wool growth.