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Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Genetic mutations cause various hair diseases, and whole genome sequencing may reveal more about these conditions.

The research found genes that may protect certain scalp cells from hair loss.

The new treatment was safe and may effectively treat male pattern hair loss.

The treatment increased hair follicle counts and is potentially effective for male hair loss without serious side effects.

The new baby shampoo and wash with stearic acid is safe and potentially effective for nourishing babies' skin.

The OVOL1-OVOL2 axis is important for hair follicle differentiation and can help diagnose certain hair-related tumors.

The symposium showed that stem cells are key for understanding and treating skin diseases and for developing new skin models and therapies.

New findings suggest potential treatments for melanoma, hyperpigmentation, hair defects, and multiple sclerosis, and show skin microbiome changes don't cause atopic dermatitis.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

ePUKs could be valuable for regenerative medicine due to their wound healing abilities.

A 72-year-old man was diagnosed with a rare skin form of Rosai-Dorfman disease after years of misdiagnosis.

Lgr5 is a marker for active, long-lasting stem cells in mouse hair follicles.

Hair follicle development is controlled by interactions between skin tissues and specific molecular signals.

β-catenin in the dermal papilla is crucial for normal hair growth and repair.

The circadian clock affects skin stem cell behavior, impacting aging and cancer risk.

Epithelial-mesenchymal interactions control hair growth cycles through specific molecular signals.

Wnt1/βcatenin signaling is crucial for heart repair after injury.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Certain mutations in a hair growth-related gene cause a type of genetic hair loss.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

Modulating BMP activity changes the number, size, shape, and type of ectodermal organs.

Macrophages help regrow hair by activating stem cells using AKT/β-catenin and TNF.

Hair, teeth, and mammary glands develop similarly at first but use different genes later.

Laminin-511 is crucial for early hair growth and maintaining important hair development signals.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

Blocking β-catenin in skin cells improves hair growth during wound healing.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Follistatin helps hair growth and cycling, while activin prevents it.