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The African spiny mouse heals skin without scarring due to different protein activity compared to the common house mouse, which heals with scarring.

Mitochondrial therapy and platelet-rich plasma therapy both stimulated hair regrowth in aging mice, with mitochondrial therapy showing similar effectiveness to plasma therapy.

Hair, teeth, and mammary glands develop similarly at first but use different genes later.

Applying calreticulin can speed up wound healing in diabetics.

Cells treated with Wnt-10b can grow hair after being transplanted into mice.

Parthenolide promotes hair growth in mice and may influence pathways related to male pattern baldness.

Bioluminescence imaging can track hair follicle cells and help study hair regrowth.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

Skin bacteria, specifically Staphylococcus aureus, help in wound healing and hair growth by using IL-1β signaling. Using antibiotics on skin wounds can slow down this natural healing process.

Different problems with hair stem cell renewal can lead to hair loss.

GLI2 increases follistatin production in human skin cells.

Glutamine metabolism affects hair stem cell maintenance and their ability to change back to stem cells.

Chemokine signaling is important for hair development.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Nerves are crucial for the regeneration of various body parts in many animals.

High levels of the protein Flightless I worsen ulcerative colitis symptoms in mice.

Foxi3 expression in developing teeth and hair is controlled by the ectodysplasin pathway.

Researchers found genetic mutations causing hypohidrotic ectodermal dysplasia in 88% of studied patients and identified new mutations and genetic variations affecting the disease.

The study found genetic differences related to hair development that may explain hair loss in a patient with Trichorhinophalangeal syndrome type I.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

Increasing Rps14 helps grow more inner ear cells and repair hearing cells in baby mice.

WWP2 is crucial for tooth development in mice.

New patents show progress in developing drugs targeting the Wnt pathway for diseases like cancer and hair loss.

Overactive Wnt signaling in mouse skin stem cells causes acne-like cysts and shrinking oil glands, which some treatments can partially fix.

Cells with active Wnt signaling are less likely to turn into cancer when exposed to a cancer-causing gene.

The Wnt/β-catenin pathway is important for tissue development and has potential in regenerative medicine, but requires more research for therapeutic use.

Wnt signaling is important for development and cell regulation but can cause diseases like cancer when not working properly.