Search

everythinglearnresearchcommunity

for

Search:↓

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Vitamin D Receptor is crucial for hair follicle shrinkage and cell death, affecting hair growth.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Non-liganded Vitamin D Receptor is crucial for healthy skin and hair.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Lithocholic acid helps hair growth and regeneration in alopecia by activating vitamin D receptors.

1,25-(OH)2D3 helps hair grow by blocking certain harmful signals.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Vitamin D receptor helps control hair growth genes in skin cells.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

People with alopecia areata often have lower vitamin D levels, which are linked to more severe and longer-lasting hair loss, but vitamin D receptor levels in the skin don't show the same pattern and don't predict treatment success.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Vitamin D receptor is crucial for starting hair growth after birth.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.