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Alopecia areata involves persistent gene abnormalities and immune activity, even in regrown hair, suggesting a risk of relapse.

Staphylococcus epidermidis A/C strains are more antibiotic-resistant and infection-adapted, while B strains thrive in hair follicles.

Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

Tissue environment greatly affects the unique epigenetic makeup of regulatory T cells, which could impact autoimmune disease treatment.

Four specific genes are linked to keloid formation and could be potential treatment targets.

Genetic analysis of rabbits identified key genes for traits like coat color, body size, and fertility.

Machine learning can predict how well patients with alopecia areata will respond to certain treatments.

The study found that sweat glands contain different types of stem cells that help with healing and maintaining healthy skin.

Researchers found 71 genetic regions linked to male pattern baldness, which account for 38% of its genetic risk.

Genomic approach finds new possible treatments for hair loss.

Pig skin cells can turn into mesodermal cells but lose their ability to become neural cells.

Macrophages help heal nerves by aiding the maturation of Schwann cells and are important for nerve repair.

YAP and TAZ proteins are necessary for the development of two types of skin cancer.

Epidermal stem cells have various roles in skin beyond just maintenance, including forming specialized structures and aiding in skin repair and regeneration.

Neurotrophin-4 increases calcium current in specific mouse neurons through the PI3K pathway.

A new technology showed that the SOX9 gene might control heart scar formation after injury, suggesting new treatment possibilities.

β-catenin is essential for stem cell activation and proliferation in hair follicles.

The research suggests that autophagy-related genes might play a role in causing alopecia areata.

Non-coding RNAs help control hair growth in cashmere goats.

SOX9 helps determine stem cell roles by interacting with DNA and proteins that control gene activity.

Schizophrenia risk genes may affect early brain development, contributing to the disease.

Enterococcus faecalis delays wound healing by disrupting cell functions and creating an anti-inflammatory environment.

Verteporfin treatment in mice led to complete skin healing without scarring.

Sex hormones may be linked to inflammation in Hidradenitis Suppurativa.

Androgenetic alopecia involves immune cell disruptions, especially increased CD4+ T cells around hair follicles.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

Lactate production is important for activating hair growth stem cells.

Ruxolitinib effectively regrows hair in most patients with severe hair loss.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.