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Research 30 of 219
- dsRNA Released by Tissue Damage Activates TLR3 to Drive Skin Regeneration
- The L412F variant of Toll-like receptor 3 (TLR3) is associated with cutaneous candidiasis, increased susceptibility to cytomegalovirus, and autoimmunity
- Noncoding dsRNA induces retinoic acid synthesis to stimulate hair follicle regeneration via TLR3
- Hypothesis: Wound‐induced TLR3 activation stimulates endogenous retinoic acid synthesis and signalling during regeneration
- Activation of TLR3 Enhance Stemness and Immunomodulatory Properties of Periodontal Ligament Stem Cells (PDLSCs)
- TLR3 Activation Stimulates Autophagy Gene Expression in Keratinocytes
- TLR3 signaling modulates immunomodulatory property of human periodontal ligament stem cells.
- Three-dimensional gelatin sponge culture potentiates MSC secretome to enhance full-thickness wound healing and induce hair follicle neogenesis via Wnt/β-catenin and TLR3/STAT3 activation in rats
- Toll-Like Receptor Function in Acute Wounds
- Through the lens of hair follicle neogenesis, a new focus on mechanisms of skin regeneration after wounding
- Altered expression of intracellular Toll-like receptors in peripheral blood mononuclear cells from patients with alopecia areata
- Toll-like Receptor 3 Agonist, Polyinosinic-polycytidylic Acid, Upregulates Carbonic Anhydrase II in Human Keratinocytes
- A Population of Radio-Resistant Macrophages in the Deep Myenteric Plexus Contributes to Postoperative Ileus Via Toll-Like Receptor 3 Signaling
- Neutrophil extracellular traps impair regeneration
- 1410 Non-coding double stranded RNA induces retinoic acid synthesis and retinoid signaling to control regeneration
- 850 After skin wounding, noncoding dsRNA coordinates prostaglandins and WNT7b to promote regeneration
- Abstract 151
- Abstract 152
- The pattern recognition receptor toll-like receptor 3 regulates skin barrier homeostasis
- 1452 Noncoding dsRNA stimulates exosome biosynthesis to regulate skin regeneration
- TO IDENTIFY THE EXPRESSION OF INTRACELLULAR TOLL-LIKE RECEPTORS (TLRS) IN PERIPHERAL BLOOD MONONUCLEAR CELLS OF ALOPECIA AREATA
- RNase L represses hair follicle regeneration through altered innate immune signaling
- RNase L represses hair follicle regeneration through altered innate immune signaling
- 1446 RNase L acts as a regeneration suppressor
- 944 Non-coding double stranded RNA induces retinoic acid synthesis and retinoid signaling to control regeneration
- Toll-like receptor-targeted anti-tumor therapies: Advances and challenges
- RNA signaling in cellular plasticity during homeostasis and regeneration
- Modulatory role of vitamins A, B3, C, D, and E on skin health, immunity, microbiome, and diseases
- 947 Accelerating wound healing mechanisms with cold atmospheric plasma
- Ca <sup>2+</sup> -mediated protein citrullination regulates proliferation in the regenerating and malignant CNS