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Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

Looking older on the outside might be linked to aging faster on the inside and can be affected by lifestyle choices and health risks.

Maintaining DNA health in stem cells is key to preventing aging and tissue breakdown.

Lamins are vital for cell survival, organ development, and preventing premature aging.

Skin aging is caused by stem cell damage and can potentially be delayed with treatments like antioxidants and stem cell therapy.

Low selenium levels can extend lifespan but worsen health issues.

Air pollution (PM10) increases skin inflammation and aging by reducing collagen and may trigger a repair response in skin cells.

The document concludes that a deeper understanding of skin aging and photodamage is needed to create better skin treatments.

Telomere damage affects skin and hair follicle stem cells by messing up important growth signals.

Hair gets thinner, grayer, and changes texture with age due to genetics, environment, and cellular changes, affecting the growth cycle.

Genetically-altered adult stem cells can help in wound healing and are becoming crucial in regenerative medicine and drug design.

Hair follicle studies suggest that maintaining telomere length could help treat hair loss and graying, but it's uncertain if mouse results apply to humans.

2011 dermatological research found new skin aging markers, hair loss causes, skin defense mechanisms, and potential for new treatments.

Tranexamic acid reduces skin aging more in female mice.

Fibroblast-derived growth factors and exosomes can significantly improve skin aging.

Progeroid mouse models show signs of early aging similar to humans, helping us understand aging better.

Deleting the Trf1 protein in mice is safe and may help prevent cancer without major side effects.

Ayurvedic herbs may reduce side effects and improve effectiveness of cancer treatments.

Short telomeres contribute to aging and cancer, and while telomerase can delay aging, it may also promote cancer.

The mouse hairy ears mutation causes longer ear hair due to changes in gene expression.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

A new ethical skin model using stem cells offers a reliable alternative for dermatological research.

Genetics, stress, and health issues can cause early hair greying, which affects self-esteem, and there's no cure, only hair dye.

Using polyethylenimine-DNA to deliver the hTERT gene can stimulate hair growth and may be useful in treating hair loss, but there could be potential cancer risks.

New treatments targeting skin stem cells show promise for skin repair, anti-aging, and cancer therapy.

Melanoma's complexity requires personalized treatments due to key genetic mutations and tumor-initiating cells.

KLF4 is important for maintaining stem cells and has potential in cancer treatment and wound healing.

Light affects skin health, aging, and cancer risk, and new light-based treatments and imaging are promising for skin care.

New treatments for skin damage from UV light using stem cells and their secretions show promise for skin repair without major risks.

PRP may help with aging and osteoarthritis, improving tissue repair and reducing surgery risk.