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A mother and son both had ulcerative colitis and alopecia, suggesting a genetic link in autoimmune disorders and successful treatment with cyclosporine.

The N363S gene variant does not cause higher adrenal androgen levels in women with polycystic ovary syndrome.

Over 67 genes linked to ichthyosis help improve diagnosis and treatment.

Women with greater androgen sensitivity respond better to finasteride for hair loss.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Maize hybrids show better early growth due to complex gene interactions from their parent strains.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

Longer TA repeats in the SRD5A2 gene may increase acne risk in Chinese people.

5α reductase type 2 enzyme mutation and oxidative stress may increase androgenetic alopecia risk in Egyptians.

Some human genetic markers work for genetic studies in pig-tailed and stump-tailed macaques, which can help in their conservation.

The document concludes that more research is needed to reduce frequent hospital visits, addiction medicine education improves with specific training, early breast cancer surgery findings are emerging, nipple smears are not very accurate, surgery for older melanoma patients doesn't extend life, a genetic condition in infants can often be treated with one drug, doctors are inconsistent with blood clot medication, a certain gene may protect against cell damage, muscle gene overexpression affects many other genes, and some mitochondrial genes are less active in mice with tumors.

A genetic mutation in the LIPH gene causes tightly curled hair that stops growing in some Japanese individuals.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

RXRα is crucial for proper immune response and links diet to immune function.

IL-4 gene variation may increase the risk of alopecia areata in Turkish people.

Melanogenesis-related proteins may trigger immune responses in alopecia areata patients.

A specific gene variation is linked to a higher risk of polycystic ovary syndrome in Caucasian women.

JAK inhibitors like tofacitinib may effectively treat Alopecia Areata.

Alopecia areata can be transferred through stem cell transplants from affected siblings.

The research found that male pattern hair loss is mostly genetic and involves hair thinning due to hormonal effects and changes in gene expression.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Certain genetic markers, especially the MICA gene, are linked to alopecia areata.

Keratosis pilaris is often linked to genetic mutations and causes skin and hair abnormalities, regardless of those mutations.

FOXN1 mutations cause severe immunodeficiency, hair loss, nail issues, and thymus defects.

Certain IL-18 gene variations may increase the risk of alopecia areata.

Alopecia areata is an autoimmune disease with genetic links, treatable with certain medications, and can affect mental health.

Scientists are still unsure what triggers the immune system to attack hair follicles in Alopecia areata.

Hidradenitis Suppurativa has genetic links, with certain gene mutations more common in patients and a third of cases having a family history.

The HSD3B1 variant increases hair loss risk in overweight women with PCOS.