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Minoxidil activates hair growth by being sulfated by P-PST in the human liver.

Liver enzyme helps minoxidil work better for blood vessel relaxation.

Adding a small amount of TPGS to minoxidil can help hair growth, but too much TPGS reduces this effect and increases minoxidil in the blood.

Human platelets change minoxidil to minoxidil sulfate, helping blood vessels widen.

Minoxidil sulfotransferase is a marker of keratinocyte differentiation and may play a role in hair growth.

Different species and human skin models vary in their skin enzyme activities, with pig skin and some models closely matching human skin, useful for safety assessments and understanding the skin's protective roles.

Minoxidil breakdown varies by enzymes, affecting hair loss treatment effectiveness.

Minoxidil boosts hair growth by undergoing a process in hair follicles and certain skin cells, and by increasing the production of compounds essential for hair growth and maintenance.

Androgen conjugates might be better indicators of skin sensitivity to hormones in women with excessive hair growth.

The enzyme PST is found in developing human kidneys and helps with detoxification and development.

Cyproterone acetate may cause liver cancer at high doses, but is considered safe at recommended doses for approved uses.

Minoxidil is less effective on eyebrows and eyelashes than scalp hair because these areas have lower enzyme activity needed to activate the drug.

Sulfotransferase SULT1A1 activity may predict minoxidil treatment success for hair loss.

Minoxidil works in liver and outer hair root sheath for hair growth.

Some plants like spinach, broccoli, and matcha may boost the effectiveness of the hair growth drug minoxidil.

Minoxidil helps hair growth by activating enzymes in hair follicles.

Minoxidil needs activation to work, and minoxidil sulfate helps with hair growth and blood pressure.

Finasteride effectively lowers specific hormone levels, helping monitor treatment progress.

DHT affects bone growth by altering gene activity in osteoblasts, potentially complicating steroid use.

The document concludes that patient outcomes for Congenital Adrenal Hyperplasia are often not ideal because of poor management and a need for better diagnosis and treatment methods.

Coumarin derivatives show promise as anti-estrogenic agents for treating breast cancer, with some in clinical trials.

Epithelial-mesenchymal interactions control hair growth cycles through specific molecular signals.

Cholesterol sulfate buildup due to a genetic mutation disrupts the skin barrier, leading to the scaling skin seen in X-linked ichthyosis.

Human enzymes can detoxify harmful substances but might also increase their cancer risk.

CAH is a genetic disorder affecting cortisol production and causing hormonal imbalances, with treatment and diagnosis varying by form and symptoms.

Low-level laser therapy can improve hair density in people with androgenic alopecia, but more research is needed to find out who it works best for.

The study developed a method to detect minoxidil and its sulfate form, found that minoxidil sulfate is temperature-sensitive, and identified a way to potentially increase its stability for hair loss treatment.

Men and women experience skin aging differently due to changes in sex hormone levels with age.

The enzyme steroid sulfatase is linked to breast cancer and other conditions, and inhibitors are being developed for treatment.

Dehydroepiandrosterone (DHEA) is a prohormone important for producing sex steroids and has potential health benefits.