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Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

GLI1 might protect against the start of skin cancer and is not linked to cancer severity.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Putting thymidine dinucleotide on newborn mice's skin can delay and reduce skin cancer.

CD4+ skin cells may be precursors to basal cell carcinoma.

GLI2 increases follistatin production in human skin cells.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

The treatment was ineffective in humans.

Vitamin D and its receptor may help prevent skin cancer.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Understanding where cancer cells come from helps create better prevention and treatment methods.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

A woman with a new PTCH gene mutation has both Gorlin syndrome and severe hair loss.

Targeting ornithine decarboxylase can help prevent skin cancer.

Stopping S100A3 activity slows down hair growth in mice.

Inhibiting ornithine decarboxylase may help prevent certain skin cancers.

Odontogenic keratocysts are caused by abnormal Hedgehog signaling and can lead to tooth and bone issues.

LDE225 is a promising skin-applied treatment for basal cell carcinoma with good skin penetration and effectiveness.

Baby teeth stem cells can help grow hair in mice.

Sulforaphane from broccoli can help protect skin from sun damage.

Intu gene is crucial for hair follicle formation by helping keratinocytes differentiate through primary cilia.

Skin needs dermal β-catenin activity for hair growth and skin cell multiplication.

Indian Hedgehog helps control skin cell growth and protects against aggressive skin cancer.

Macrophages are vital for skin healing, hair growth, salt balance, and cancer defense.

The topical inhibitor CUR61414 was not effective in treating basal cell carcinoma in human trials.

Exosomes from human skin cells can stimulate hair growth and could potentially be used for treating hair loss.

Brg1 is crucial for hair growth and skin repair by maintaining stem cells and promoting regeneration.

The article concludes that hair loss is a common side effect of drugs treating skin cancer by blocking the hedgehog pathway, but treatment should continue, and more selective drugs might prevent this side effect.