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The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

The mTOR signaling pathway is crucial for hair health and targeting it may lead to new hair loss treatments.

Astilbin can potentially calm overactive immune responses, like in Type 1 Diabetes, by suppressing certain cell activities and reducing inflammation.

PPAR-γ may be a key target for treating alopecia areata and other skin conditions.

New understanding of the causes of primary cicatricial alopecia has led to better diagnosis and potential new treatments.

Lysophospholipids like LPA and S1P are important for hair growth, immune responses, and vascular development, and could be targeted for treating diseases.

The symposium concluded that a specific lipid might cause hair loss, inflammation is a key factor, and stem cells in bald scalps aren't working right, suggesting new treatment targets.

Paneth cells and intestinal stem cells work together metabolically for stem cell function and regeneration.

Multi-omics techniques help understand the molecular causes of androgenetic alopecia.

Low doses of mixed chemicals cause permanent reproductive malformations in male rats.

The review suggests that while many AKR1C3 inhibitors show promise for treating certain cancers, more research is needed to confirm their effectiveness in humans.

SCD1 is crucial for skin health and overall energy balance.

Lipocalin-Type Prostaglandin D2 Synthase (L-PGDS) is a protein that plays many roles in the body, including sleep regulation, pain management, food intake, and protection against harmful substances. It also affects fat metabolism, glucose intolerance, cell maturation, and is involved in various diseases like diabetes, cancer, and arthritis. It can influence sex organ development and embryonic cell differentiation, and its levels can be used as a diagnostic marker for certain conditions.

Higher miR-34a levels and the A variant of the MIR-34A gene are linked to increased risk and severity of alopecia areata.

Finasteride harms Daphnia magna's reproduction, growth, and metabolism.

Curcumin supplements increase adiponectin and decrease leptin in adults.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

FGF signaling is a promising target for developing treatments for wounds, metabolic diseases, and cancer.

Substance P may worsen acne by increasing inflammation, but corticosteroids might help by reducing this effect.

Blocking EGFR in skin cells doesn't majorly increase inflammation markers.

Retinoic acid-binding proteins in skin are regulated by β-catenin and Notch signalling.

Eating a high-fat fish oil diet caused mice to lose hair due to a specific immune cell activity in the skin linked to a protein called E-FABP.

Pioglitazone usually doesn't effectively treat or cure lichen planopilaris.

Taking oral isotretinoin with creams worked better for treating a type of hair loss than creams alone.

AFM can help diagnose lichen planopilaris by identifying specific hair structure changes.

Protein profiling of forehead skin can help distinguish between frontal fibrosing alopecia and androgenetic alopecia.

TGFβ signaling prevents sebaceous gland cells from producing fats.

Lysophosphatidic acid affects sensory neurons and may cause neuropathic pain and itch.

Some wound-healing cells can turn into fat cells around new hair growth in mice.