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Feather growth and regeneration involve complex patterns, stem cells, and evolutionary insights.

Researchers found a genetic link for hereditary hair loss but need more analysis to identify the exact gene.

A specific chemical change in the S100A3 protein leads to the formation of a four-part structure important for hair formation.

Notch signalling helps skin cells differentiate and prevents tumors.

EGFR-TKIs can cause significant skin, nail, and organ side effects.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Targeting androgen receptors could be a promising way to treat skin disorders with fewer side effects.

The conclusion is that Pigmented Epithelioid Melanocytoma can start from hair follicle stem cells or from a mole on the skin.

Vitamin D is important for bones, hair, blood pressure, and breast development.

Prostaglandin D₂ might be targeted for new male pattern baldness treatments.

Celsr1 is crucial for skin cell alignment, while Celsr2 has little effect on this process.

Some viruses can cause cancer by changing cell processes and avoiding the immune system; vaccines and targeted treatments help reduce these cancers.

Skin side effects from EGFR inhibitor cancer treatment can be managed effectively, often without stopping the medication.

Fibroblast growth factor receptor signaling controls cell development and repair, and its malfunction can cause disorders and cancer, but it also offers potential for targeted therapies.

Understanding and manipulating epigenetic changes can potentially lead to human organ regeneration therapies, but more research is needed to improve these methods and minimize risks.

NF-κB signaling is crucial in many diseases and can be targeted for new treatments.

Different processes create patterns in skin and things like hair and feathers.

A specific gene change plus an additional mutation in the same gene cause hereditary trichilemmal cysts.

Cystatin M/E strongly inhibits cathepsin V and cathepsin L, important for skin formation.

Hair follicles in the back of the rosette fancy mouse have reversed orientations due to a gene mutation.

New treatments for advanced skin cancer are improving patient outcomes, but drug resistance and finding the right treatment combinations are still big challenges.

The immune system plays a key role in tissue repair, affecting both healing quality and regenerative ability.

Restoring mitochondrial function in mice reversed their skin wrinkling and hair loss.

MAD2B slows down the growth of skin cells that are important for hair development by interacting with TCF4.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Foxn1 gene regulation is crucial for thymus development but not for hair growth.

Tiny particles from 3D-grown skin cells speed up wound healing by promoting blood vessel growth.

Using radiation to make mice's hair turn gray helps study and find ways to prevent or reverse hair graying.

Mutations in iRhom2 affect hair and skin in mice and are linked to esophageal cancer, with ADAM17 playing a crucial role.

Prostasin has two roles in skin: one for normal skin development without needing activation, and another for proper hair growth that requires activation.