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Hair grows faster in the morning and is more vulnerable to damage from radiation due to the internal clock in hair follicle cells.

Mice genetically modified to produce more Del1 protein had faster hair regrowth.

Mice without the p21 gene can fully regenerate injured ears due to reduced Sdf1 increase and leukocyte recruitment, suggesting new ways to induce tissue regeneration in mammals.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Mice with human chymase had a higher death rate when exposed to a toxin compared to normal mice.

Regulatory T cells are essential for normal and improved wound healing in mice.

Too much zinc in the diet can cause hair loss and color change in young mice by reducing copper in the body.

An imbalanced gut and lack of biotin can cause hair loss in mice.

Obese mice with a leptin gene mutation have a longer resting phase in their hair cycle, which may help understand certain hair loss conditions.

SV40 T antigen in hair follicles causes abnormal hair and health issues in mice.

Mice are useful for researching human hair loss and testing treatments, despite some differences between species.

Human stem cells can help form hair follicles in mice.

Ginkgo biloba leaf extract may help hair regrowth and improve heart health.

The Naked (N) trait in mice is linked to lower glycine and tyrosine in hair proteins.

Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

The gene Foxq1, controlled by Hoxc13, is crucial for hair follicle differentiation.

A protein from certain immune cells is key for new hair growth after skin injury in mice.

Cathepsin L is essential for normal hair growth and development.

P-selectin is not the only factor that prevents scarring in fetal wound healing in mice.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

M30 is a promising treatment for preventing hair loss during chemotherapy.

Lack of sleep in mice leads to prostatitis by reducing certain hormones and activating an inflammatory pathway, which can be temporarily fixed with normal sleep.

Hairless mice lack fur due to a genetic mutation affecting skin response, not hormone issues.

Mice with alopecia areata had wider lymphatic vessels in their skin.

The Naked gene in mice causes abnormal sebaceous glands and disrupts hair follicle organization.

Mutant mice help researchers understand hair growth and related genetic factors.

Deleting the Far2 gene in mice causes sebaceous gland issues and patchy hair loss.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Insulin helps heal corneal wounds and nerves in diabetic mice by activating the Wnt signaling pathway.

Mice with autoimmune hair loss showed signs of heart problems.