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Certain genes are more active during wound healing in axolotl and Acomys, which could help develop materials that improve human wound healing and regeneration.

Fibroblast changes in systemic sclerosis may help understand disease severity and treatment.

Autophagy helps keep skin healthy and may improve treatments for skin diseases.

Thy-1 protein helps improve blood flow and wound healing in the skin.

Hedgehog signaling in skin cells is crucial for hair growth and skin healing, but needs to be balanced to avoid harmful effects like scarring and cancer.

Fat cells in the skin help start healing and form important repair cells after injury.

AMT may cause hair loss and changing dWAT activity could help treat it.

Faulty inflammasome activation may lead to autoimmune skin diseases and could be a target for new treatments.

Platelet-rich plasma (PRP) speeds up skin wound healing and has potential in medical and cosmetic uses.

Blocking DPP4 can potentially speed up hair growth and regeneration, especially after injury or in cases of hair loss.

Fibromodulin treatment helps reduce scarring and improves wound healing by making it more like fetal healing.

The gel with icariin speeds up wound healing, reduces scarring, and helps hair growth by controlling BMP4 signaling. It also reduces inflammation and improves wound quality in mice, adapts to different wound shapes, and gradually releases icariin to aid healing. It also prevents too much collagen and myofibroblast formation during skin healing.

Integrin alphavbeta6 is important for wound healing and hair growth, and blocking it may improve these processes.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Activating TRPV4 in skin cells helps regrow hair in mice, possibly offering a treatment for hair loss.

Wnt signaling is crucial for skin wound healing and reducing scars.

PDGF signaling is crucial for maintaining fat stem cells in the skin, and its level of activation can either preserve these cells or cause fibrosis.

The PDGF/PDGFR pathway is a potential drug target with mixed success in treating various diseases, including some cancers and fibrosis.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.

Inactive hair follicle stem cells help prevent skin cancer.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Tsukushi helps control inflammation and aids in wound healing.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

ILK and ELMO2 help cells move and stick together, important for wound healing and hair growth.

Notch1 signaling is crucial for improving wound healing and skin regeneration by affecting stem cell behavior.

CD201+ fascia progenitors are essential for wound healing and could be targeted for treating skin conditions.

Using developmental signaling pathways could improve adult wound healing by mimicking scarless embryonic healing.

Aged individuals heal wounds less effectively due to specific immune cell issues.

P144® improves hypertrophic scars by reducing size and thickness and increasing elasticity.

MSCs and their exosomes may speed up skin wound healing but need more research for consistent use.