Search

everythinglearnresearchcommunity

for

Search:↓

The research found that Atypical Progeroid Syndrome has unique symptoms and is not caused by the buildup of a certain mutant protein.

Thujae occidentalis semen extract may help treat male pattern baldness by blocking a hair loss-related enzyme and reducing hair loss in mice.

Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.

Activating Wnt in skin cells controls the number of hair follicles by directing cell movement and fate.

Mutant cells in hair follicles are influenced by their location and interactions with surrounding cells.

Mouse hair follicle cells briefly grow during the early hair growth phase, showing that these cells are important for starting the hair cycle.

Hair patterns in mice are controlled by both a global system dependent on Fz6 and a local self-organizing system.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

The method quickly analyzes hair growth genes and shows that blocking Smo in skin cells stops hair growth.

Mice with a virus similar to COVID-19 had skin damage, but a special treatment helped repair it.

Hair grows when stem cell offspring in the follicle base proliferate, influenced by the dermal papilla.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Targeting androgen receptors could be a promising way to treat skin disorders with fewer side effects.

Phospholipase A2 is necessary for the correct placement of PIN proteins in plant roots, affecting root growth.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

Topical tofacitinib is effective in promoting hair growth for non-scarring alopecia.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

Bezafibrate treatment improved skin and spleen health in aging mice but didn't extend lifespan.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

Disrupted stem cell signals in hairpoor mice cause hair loss.

MILL molecules are unique immune proteins in mice that don't need TAP to appear on cell surfaces.

Isoallopregnanolone may be a safe and effective treatment for reducing tics in a mouse model of Tourette syndrome.

Hair follicles can regrow in wounded adult mouse skin using a process like embryo development.

Basal cell carcinoma mostly starts from cells in the upper skin layers, not hair follicle stem cells.

Scientists found a specific group of itch-sensing nerve cells in mice important for feeling itch but not for sensing heat or touch.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.