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Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Deleting the CRIF1 gene in mice disrupts skin and hair formation, certain proteins affect hair growth, a new compound may improve skin and hair health, blood cell-derived stem cells can create skin-like structures, and hair follicle stem cells come from embryonic cells needing specific signals for development.

The study found that bioengineered hair follicles work when using cells from the same species but have issues when combining human and mouse cells.

Adding a specific gene to skin cells can help treat skin disorders like psoriasis.

The patch assay can create mature hair follicles from human cells and may help in hair loss treatments.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

Epigenetic changes control how adult stem cells work and can lead to diseases like cancer if they go wrong.

Cyclosporin A promotes hair cell growth and affects protein kinase C levels.

SDF-1/CXCL12 and its receptor CXCR4 are crucial for melanocyte movement in mouse hair follicles.

The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

Hair follicle cells resist turning into skin cells.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Melanocyte stem cells are crucial for skin pigmentation and have potential in disease modeling and regenerative medicine.

Adding TERT and BMI1 to certain skin cells can improve their ability to create hair follicles in mice.

OCT binds strongly to hair sheath cells and may affect skin and hair growth with fewer side effects than vitamin D3.

Skin cells and certain hair follicle areas produce hemoglobin, which may help protect against oxidative stress like UV damage.

Nonpalmoplantar skin cells can be made to express keratin 9 by interacting with palmoplantar fibroblasts.

A specific group of skin stem cells was found to help maintain hair follicle cells.

New cell-based therapies may improve hair loss treatments in the future.

The engineered skin substitute helped grow skin with hair on mice.

Scientists created three types of structures to help regrow hair follicles, and all showed promising results for hair regeneration.

DNA methylation is essential for skin and hair follicle development, and could be a target for treating skin diseases.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Putting α-methylspermidine on mouse skin can start hair growth.

Fully regenerating human hair follicles not yet achieved.

Changes in keratin affect skin health and can lead to skin disorders like blistering diseases and psoriasis.

Celsr1 is crucial for skin cell alignment, while Celsr2 has little effect on this process.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.