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Midscalp hair density and terminal hair percentage are good indicators of female pattern hair loss severity.

Higher CD70 and CD27 gene expression in alopecia areata lesions predicts disease severity and activity.

Injecting monocyto-angiotropin into hare skin increases hair growth by forming new blood vessels.

hsa-miR-193a-5p may help diagnose and treat alopecia areata.

Low HDL-C, uric acid, and 25-hydroxyvitamin D are risk factors for early-onset male hair loss.

Canine pemphigus foliaceus involves significant immune activity and shares similarities with human pemphigus.

Women with Polycystic Ovary Syndrome are more likely to have Non-Alcoholic Fatty Pancreas Disease, which is associated with older age, metabolic syndrome, insulin resistance, and high male hormone levels.

TSC2 is crucial for proper hair follicle development and patterning.

Immune activities and specific genes are important in male pattern baldness.

Higher levels of heat shock protein 27 and lower levels of miR-1 can increase AR levels, leading to hair loss in men.

Higher serum androgens contribute to male pattern baldness.

Androgenetic alopecia is a long-term, immune-related disorder that starts during puberty due to androgen secretion, and it might be improved with iron tablets, platelet transfusion, and anti-inflammation therapy.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Alopecia areata patients have more Propionibacterium acnes and less Staphylococcus epidermidis on their scalps.

Alopecia areata patients show unique protein activity patterns, suggesting imbalanced signaling pathways.

Bioengineering can potentially treat hair loss by regenerating hair follicles and cloning hair, but the process is complex and needs more research.

T-cells in alopecia areata scalp show abnormal regulation, leading to less inflammation.

Higher levels of ischemia-modified albumin in telogen effluvium patients may indicate oxidative stress.

Certain microRNAs might help identify and understand Frontal Fibrosing Alopecia.

A specific gene variation is linked to a higher risk of polycystic ovary syndrome in Caucasian women.

The exact causes of baldness are not fully understood, limiting treatment options.

TDM10842, a thyroid hormone receptor activator, was found to effectively promote hair growth in mice.

Certain gene variations may increase the risk of hair loss in Egyptians.

Experts met to improve care for ichthyosis patients in Spain.

AFM can help diagnose lichen planopilaris by identifying specific hair structure changes.

Plant extracts may help prevent or reverse hair graying.

Certain gene variations in PITX2 are linked to a higher risk of male pattern baldness in Indians.

Hormonal imbalances are important in the development of male pattern baldness.

The study suggests that changes in immune system receptors and their interaction with a cell component may be important in the development of a type of hair loss condition.

People with alopecia have shorter hair follicles and more c-kit, a stem cell factor receptor, which could predict how the condition progresses.