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Overexpression of SSAT causes hair loss and skin issues, but reducing putrescine can help.

K42 and K124 keratins are only found in horse hoof lamellae.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

Hair follicle stem cells in hairpoor mice are disrupted, causing hair loss.

Targeting ornithine decarboxylase can help prevent skin cancer.

The study found new details about human hair growth and suggests that preventing a specific biological pathway could potentially treat hair graying.

High ODC and low K1 and K10 may indicate early skin tumors in mice.

The review highlights the importance of stem cells in hair health and suggests new treatment strategies for hair loss conditions.

Putting α-methylspermidine on mouse skin can start hair growth.

Fatty acid transport protein 4 is essential for skin and hair development.

Overexpressing ornithine decarboxylase and v-Ha-ras in keratinocytes leads to invasiveness and malignancy.

Activating Nrf2 in skin cells causes skin disease similar to chloracne in mice.

Overexpression of activin A in mice skin causes skin thickening, fibrosis, and improved wound healing.

Different types of stem cells and their environments are key to skin repair and maintenance.

The conclusion is that skin and hair patterns are formed by a mix of cell activities, molecular signals, and environmental factors.

Certain human skin cells marked by CD44 and ALDH are rich in stem cells capable of long-term skin renewal.

A protein called desmoglein 3 is important for keeping hair follicle stem cells inactive and helps in their regeneration.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

The document concludes that understanding the sebaceous gland's development and function is key to addressing related skin diseases and aging effects.

The document suggests a bacteria plays a significant role in acne rosacea and that white hair can regain color after transplant, meriting more research on reversing grey hair.

Hormones and stretching both needed for nipple area skin growth in mice.

Proteins like aPKC and PDGF-AA, substances like adenosine and ATP, and adipose-derived stem cells all play important roles in hair growth and health, and could potentially be used to treat hair loss and skin conditions.

Hair growth environment recreated with challenges; stem cells make successful skin organoids.

Hair thinning causes stem cell loss through a process involving Piezo1, calcium, and TNF-α.

Smad1 and Smad5 are essential for hair follicle development and stem cell sleepiness.

Epidermolysis bullosa simplex causes easily blistered skin due to faulty skin cell proteins, leading to new treatment ideas.

New treatments targeting specific genes show promise for treating keratin disorders.

Hdac1 and Hdac2 help maintain and protect the cells that control hair growth.

Thyroid hormones are important for skin health and changes in them can affect conditions like hair loss and eczema.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.