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Inactive stem cells in hair follicles and muscles can avoid detection by the immune system.

Hair follicles help skin immune recovery after UVB exposure.

Lung and liver macrophages protect our tissues and their dysfunction can cause various diseases.

The document concludes that wound healing leads to skin repair or scar formation.

Immune cells help regulate hair growth, and better understanding this can improve hair loss treatments.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

The document concludes that managing side effects of MS therapies is crucial for treatment success and patient adherence.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Stress may trigger hair loss by affecting immune protection in hair follicles.

The immune system plays a key role in tissue repair, affecting both healing quality and regenerative ability.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Different immune cells like platelets, mast cells, neutrophils, macrophages, T cells, B cells, and innate lymphoid cells all play roles in skin wound healing, but more research is needed due to inconsistent results and the complex nature of the immune response.

Androgenetic alopecia involves immune cell disruptions, especially increased CD4+ T cells around hair follicles.

Immune activities and specific genes are important in male pattern baldness.

The document concludes that understanding how the skin's immune system and inflammation work is complex and requires more research to improve treatments for skin diseases.

TGFβ's manipulation of inflammation and immune cells affects cancer spread, suggesting new treatment strategies and biomarkers.

Alopecia areata involves immune response and gene changes affecting hair loss.

A protein called lfTSLP is important in causing allergic and other skin diseases and could be a target for treatment.

Some viruses can cause cancer by changing cell processes and avoiding the immune system; vaccines and targeted treatments help reduce these cancers.

Changes to the Foxp3 protein affect how well regulatory T cells can control the immune system, which could help treat immune diseases and cancer.

Older skin has higher cancer risk due to inflammation and stem cell issues.

CRH in the skin acts like the body's stress response system, affecting cell behavior and immune activity.

The skin's internal clock affects healing, cancer risk, aging, immunity, and hair growth, and disruptions can harm skin health.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

The document concludes that while there are various treatments for Alopecia Areata, there is no cure, and individualized treatment plans are essential due to varying effectiveness.

New methods to improve the healing abilities of mesenchymal stem cells for disease treatment are promising but need more research.

Lysophospholipids like LPA and S1P are important for hair growth, immune responses, and vascular development, and could be targeted for treating diseases.

Blocking JAK/STAT pathways can help treat hair loss from alopecia areata.

Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

New treatments targeting T-cell pathways are needed for better alopecia areata management.