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A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

Cholesterol-related compounds can stop hair growth and cause inflammation in a type of scarring hair loss.

TGFβ's manipulation of inflammation and immune cells affects cancer spread, suggesting new treatment strategies and biomarkers.

Immune cells help regulate hair growth, and better understanding this can improve hair loss treatments.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.

Botryococcus terribilis Ethanol Extract may reduce inflammation by changing gene expression in cells.

Fat cells in the skin help start healing and form important repair cells after injury.

Ingenol mebutate gel changes gene expression related to skin development and immune response in actinic keratosis.

Wild African goats have genetic adaptations for surviving harsh desert conditions.

A new mutation in the STING protein causes a range of symptoms and its severity may be affected by other genetic variations; treatment with a specific inhibitor showed improvement in one patient.

DNA methylation changes are linked to hair growth cycles in goats.

Keloid skin disorder involves abnormal fibroblast activation and immune response, linked to a group of genes including FGF11.

The study found that immune responses disrupt hair growth cycles, causing hair loss in alopecia areata.

CD2 might be a new treatment target for patchy alopecia areata.

Plasmacytoid dendritic cells are a key factor in causing hair loss in alopecia areata and could help differentiate it from other hair loss conditions.

Different immune responses cause hair loss in scalp diseases, with unique patterns in scalp psoriasis possibly protecting against hair loss.

Hair loss in male pattern baldness is linked to changes in immune cell behavior around hair follicles.

Androgenetic alopecia involves immune cell disruptions, especially increased CD4+ T cells around hair follicles.

Different types of sun exposure damage skin cells and immune cells, with chronic exposure leading to more severe and lasting damage.

Four specific genes are linked to keloid formation and could be potential treatment targets.

The telogen phase of hair growth is active and important for preparing hair follicles for regeneration, not just a resting stage.

Blood tests can help understand the genetic differences in people with alopecia areata, including how severe it is and if it's inherited.

Melanocytes may trigger the immune response in alopecia areata, affecting hair regrowth.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

Certain microRNAs are linked to various skin diseases and could be used to diagnose and treat these conditions.

MicroRNAs play a crucial role in skin and hair health, affecting everything from growth to aging, and could potentially be used in treating skin diseases.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

IL-1 family cytokines are crucial for skin defense and healing, but their imbalance can cause skin diseases.

The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.

Aged individuals heal wounds less effectively due to specific immune cell issues.