Search

everythinglearnresearchcommunity

for

Search:↓

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.

γδTregs may help treat autoimmune diseases like alopecia areata by promoting hair regrowth and reducing immune attacks.

Cell-based therapy using specific immune cells may help treat alopecia areata by promoting hair regrowth.

Stress may contribute to hair loss in alopecia areata by affecting immune responses and cell death in hair follicles.

The document concludes that more research is needed to better understand and treat primary cicatricial alopecias, and suggests a possible reclassification based on molecular pathways.

The document concludes that while there are various treatments for Alopecia Areata, there is no cure, and individualized treatment plans are essential due to varying effectiveness.

Targeting the protein Caveolin-1 might help treat a type of scarring hair loss called Frontal Fibrosing Alopecia.

Oxidative stress may contribute to hair loss in alopecia areata and antioxidants could potentially help as a treatment.

ILC1 cells contribute to hair loss in alopecia areata.

Regulatory γδ T cells help protect hair follicles from alopecia areata and promote hair regrowth.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

The new aptamer TAGX-0003 shows promise as an effective treatment for hair loss disorder alopecia areata.

Innate lymphoid cells type 1 may contribute to alopecia areata.

The conference highlighted new diagnostic tools, the role of genetics in hair loss, and emerging treatments.

New treatments targeting T-cell pathways are needed for better alopecia areata management.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

Alopecia areata, a type of hair loss, is likely an autoimmune disease with a genetic link, but its exact cause is still unknown.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

Stress hormones and autoimmune reactions can cause hair loss.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

Sorafenib may cause hair loss in a way similar to alopecia areata.

AIRE deficiency causes hair loss similar to alopecia areata in mice.

Vδ1+ T-cells in the skin contribute to hair loss in alopecia areata and could be targeted for treatment.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

New treatments for alopecia areata show promise, but standardized guidelines are needed.

Alopecia Areata is an autoimmune condition causing hair loss with no cure and treatments that often don't work well.

Treg cell-based therapies might help treat hair loss from alopecia areata, but more research is needed to confirm safety and effectiveness.

Immune cells help regulate hair growth, and better understanding this can improve hair loss treatments.