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Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

The document concludes that more research is needed on making and understanding biomaterial scaffolds for wound healing.

A vitamin D receptor mutation causes rickets and affects immune responses.

sPLA2-IIA increases growth in hair follicle stem cells and cancer cells, suggesting it could be targeted for hair growth and cancer treatment.

UV radiation may cause DNA changes in skin, certain UVB therapy helps psoriasis, a new gene mutation is linked to mild piebaldism, different immune cells affect psoriasis, a drug promotes hair growth, and some cancer drugs could treat skin barrier issues.

Vitamin D is essential for bone health and may reduce risks of several diseases.

Minoxidil boosts enzymes that help hair growth.

The spiny mouse can regenerate its skin without scarring, which could help us learn how to heal human skin better.

iPSCs can help treat genetic skin disorders by creating healthy skin cells from a small biopsy.

Activating c-Myc in skin causes rapid cell growth and changes, but these effects are reversible.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Using A132 hair dye with sunlight can cause skin damage and hair loss.

Sweat gland development involves two unique skin cell programs and a temporary skin environment.

ADAM 10 and ADAM 12 proteins are involved in different stages of hair growth and could be targets for treating hair disorders.

Oxytocin receptors are found in skin cells near touch and pain neurons.

Hair follicle pores help cell survival and growth, even after radiation.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

The document concludes that managing skin conditions during pregnancy is important and requires specialized care.

Li2CO3 improved skin disease in a mouse model of Focal Dermal Hypoplasia without toxicity.

Caffeine applied to the scalp can protect hair follicles from UV damage.

The study concluded that the new wound model can be used to evaluate skin regeneration and nerve growth.

The method increases stem-like cells for better skin regeneration.

Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

A substance called FGF9 from certain immune cells can trigger new hair growth during wound healing in mice, but humans may not have the same response due to fewer of these cells.

Certain proteins, caspases-1 and -11, are important in the early development of skin inflammation in mice.

The conclusion is that CD90 is not a specific marker for fibroblast subtypes and better methods are needed to identify them.

The document concludes that mouse models are helpful but have limitations for skin wound healing research, and suggests using larger animals and genetically modified mice for better human application.

Stem cells regenerate tissues and their behavior varies by environment, suggesting the hematopoietic system model may need revision.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

The cornified envelope is crucial for skin's barrier function and involves key proteins and genetic factors.