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A protein called PCBP2 controls the production of a hair growth protein by interacting with its genetic message and is linked to hair loss when this control is disrupted.

Mutations in the hairless gene in mice affect its expression and lead to a range of developmental issues in multiple tissues.

A new mutation in the hairless gene causes hair loss and skin wrinkling in mice.

Vitamin D receptor helps control hair growth genes in skin cells.

Mice with a changed Hr gene lose and regrow hair due to changes in the gene's activity.

The HR protein's role as a repressor is essential for controlling hair growth.

The commentary explains that a balance of HR protein and putrescine is important for normal hair growth.

A mutation in the HR gene causes a rare form of irreversible hair loss in two Kashmiri families. Whole exome sequencing is effective for finding such mutations.

Certain mutations in a hair growth-related gene cause a type of genetic hair loss.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

New and known mutations in the hairless gene cause a hair loss condition called Atrichia with papular lesions.

The Hairless gene is crucial for healthy skin and hair growth.

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Hairless protein can block vitamin D activation in skin cells.

Hairless protein and putrescine regulate each other, affecting hair growth and skin balance.

Histone demethylases play a key role in the development of many diseases and may be targets for treatment.

A new mutation in the HR gene causes hair loss in a specific family.

Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

Hairless protein affects hair follicle structure by regulating the Dlx3 gene.

Damage to skin releases dsRNA, which activates TLR3 and helps in skin and hair follicle regeneration.

Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

A new mutation in the Hairless gene causes hair loss in two Pakistani families.

Researchers found a new mutation causing total hair loss from birth.

A Taiwanese patient had hair loss and skin bumps without the usual gene mutation, suggesting other genetic factors might be involved.

Higher enzyme levels found in women with hair loss, suggesting hormone imbalance.

Increased cell death and reduced cell growth in hair follicles contribute to baldness.

Certain miRNAs may play a role in sheep hair follicle development, which could help improve wool production.