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All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Blocking β-catenin in skin cells improves hair growth during wound healing.

Mutant mice help researchers understand hair growth and related genetic factors.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Hair shedding is an active process that could be targeted to treat hair loss.

EVAL membranes help create cell structures that can regrow hair follicles.

Researchers developed a quick and easy way to get and grow cells from the base of human hair follicles.

Targeting the skin's endocannabinoid system could help treat skin disorders.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

TRH stimulates human hair growth and extends the hair growth phase.

HFMs can help study hair growth and test potential hair growth drugs.

Human hair follicles produce and respond to erythropoietin, helping protect against stress.

The p53 protein helps control hair follicle shrinking by promoting cell death in mice.

Tiny needles with valproic acid can effectively regrow hair.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

An imbalance between certain immune cells is linked to a chronic skin condition and may be influenced by obesity, smoking, and autoimmune issues.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

Prolactin affects the production of different keratins in human hair, which could lead to new treatments for skin and hair disorders.

The TRPV3 gene mutation affects hair growth by keeping mice in the growth phase longer, which could help treat hair loss.

Exosomes from human skin cells can stimulate hair growth and could potentially be used for treating hair loss.

The document concludes that while there are various treatments for Alopecia Areata, there is no cure, and individualized treatment plans are essential due to varying effectiveness.

Disruptions in hair follicle fibroblast dynamics can cause hair growth problems.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Alopecia areata shows a unique type 1 interferon signature, suggesting potential treatment by targeting this pathway.

Human skin cells can create new hair follicles when transplanted into mice.

Prolactin may play a significant role in skin and hair health and could be a target for treating skin and hair disorders.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.

Estrogens generally inhibit hair growth and improve skin quality, but their exact effects on hair follicles are complex and not fully understood.