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Research 31–60 of 1000+
- Lichen planopilaris is characterized by immune privilege collapse of the hair follicle's epithelial stem cell niche
- Hair Follicle Mesenchyme-Associated PD-L1 Regulates T-Cell Activation Induced Apoptosis: A Potential Mechanism of Immune Privilege
- Epidermal growth factor receptor/mitogen-activated kinase inhibitor treatment induces a distinct inflammatory hair follicle response that includes collapse of immune privilege
- Scarring hair follicle destruction is driven by the collapse of EGFR-protected JAK-STAT1-sensitive stem cell immune privilege
- BI02 (P97): Epidermal growth factor receptor inhibitor therapy induces a distinct inflammatory hair follicle response that includes a collapse of immune privilege
- LB789 Novel IFNγ aptamer TAGX-0003 protected hair follicles from immune privilege collapse and reversed Alopecia Areata like phenotype in humanized mouse model
- Microneedle‐mediated Delivery of Immunomodulators Restores Immune Privilege in Hair Follicles and Reverses Immune‐Mediated Alopecia
- Somatostatin Expression in Human Hair Follicles and Its Potential Role in Immune Privilege
- UVB-induced depletion of donor-derived dendritic cells prevents allograft rejection of immune-privileged hair follicles in humanized mice
- 1338 Epidermal growth factor receptor protects from immune privilege collapse and scarring hair follicle destruction
- 700 Tyrosine kinase 2 inhibition rescues hair follicles from IL-12–mediated immune privilege collapse and reverses the induction of human alopecia areata in a humanized mouse model
- 061 Selective inhibition of tyrosine kinase 2 prevents and restores interleukin-12-induced hair follicle immune privilege collapse: a novel approach to alopecia areata therapy?
- LB1777 A novel ex vivo model of human hair follicle immune privilege collapse reveals the potential of farudodstat, a DHODH inhibitor, as a therapeutic for alopecia areata treatment
- ISIDLB1777 - A novel ex vivo model of human hair follicle immune privilege collapse reveals the potential of farudodstat, a DHODH inhibitor, as a therapeutic for alopecia areata treatment
- 861 EGFR/MEK inhibitor therapy induces partial hair follicle immune privilege collapse in vivo and ex vivo
- ISID1338 - Epidermal growth factor receptor protects from immune privilege collapse and scarring hair follicle destruction
- 63418 JAK3/TEC kinase pathways are active in alopecia areata lesions, and their inhibition with Ritlecitinib prevents αCD3/αCD28+IL-2 induced immune privilege collapse in healthy hair follicles ex vivo
- 0047 Loss of transposable element silencing sensitizes hair follicles to immune privilege collapse in alopecia areata
- P97: Epidermal growth factor receptor inhibitor therapy induces a distinct inflammatory hair follicle response that includes a collapse of immune privilege
- Lessons from TNF-α Inhibitor–Induced Alopecia Areata: Does TNF-α Operate as a Hair Follicle Immune Privilege Guardian under Inflammatory Conditions?
- 0046 Antagonizing interferon-gamma receptor-1 signaling with topical SAMiRNA in human scalp skin protects hair follicles from immune privilege collapse and keratinocyte apoptosis
- Immune niches for hair follicle development and homeostasis
- Hair follicles as immune targets: unraveling cellular players, cytokine crosstalk, and precision pathways for future therapy of alopecia areata
- Engineering immune-competent hair follicle microphysiological systems: from organoid assembly to dynamic immune modelling
- 089 Neoplasms bypass immune evasion via hair follicle residence
- Probing the Effects of Stress Mediators on the Human Hair Follicle
- A neuroendocrinological perspective on human hair follicle pigmentation
- 001 Human scalp hair follicles are protected from alopecia areata in vivo by regulatory γδT cells
- 175 Understanding immune privilege in an inducible mouse model of primary cicatrcial alopecia
- Pathogenesis and regenerative therapy in vitiligo and alopecia areata: focus on hair follicle