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Personalized medicine in dermatology uses molecular biomarkers to improve diagnosis and treatment but needs further advancements for practical use.

Certain fungi protect skin health, but changes can allow harmful fungi to cause serious infections, needing more research for treatment and control.

Altering SSAT affects fat metabolism and body fat in mice.

Fibroblast changes in systemic sclerosis may help understand disease severity and treatment.

The research confirms that Hidradenitis Suppurativa is characterized by increased inflammation, disrupted skin cell organization, and abnormal metabolic processes.

Blocking certain immune signals can reduce skin damage from radiation therapy.

Reductive stress messes up collagen balance and alters cell signaling in human skin cells, which could help treat certain skin diseases.

Different ligands change the shape of the TRPV3 ion channel in unique ways.

Certain genes are more active during wound healing in axolotl and Acomys, which could help develop materials that improve human wound healing and regeneration.

Colorectal cancer development involves both genetic changes and epigenetic alterations like DNA methylation and microRNA changes.

Mutations in the RBPJ gene cause Adams-Oliver Syndrome.

Damaging mutations in NFKB2 cause a severe and distinct form of primary immunodeficiency with early-onset and often ACTH-deficiency.

Diabetes causes lasting cell dysfunctions, leading to serious complications even after blood sugar is controlled.

IL-1 family cytokines are crucial for skin defense and healing, but their imbalance can cause skin diseases.

Type 1 diabetes often occurs with other autoimmune diseases, and personalized treatment based on genetics can improve outcomes.

Nitric Oxide has potential in medicine, especially for infections and heart treatments, but its short life and delivery challenges limit its use.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Melasma's causes include genetics, sun exposure, hormones, and oxidative stress, and understanding these can help create better treatments.

Certain natural and synthetic compounds may help treat inflammatory skin diseases by targeting a specific signaling pathway.

Oxidative stress worsens PCOS by damaging cells and disrupting metabolism, suggesting antioxidant treatments might help.

Genomic research can help improve the quality and production of natural fibers in animals.

Cell polarity signaling controls tissue mechanics and cell fate, with complex interactions and varying pathways across species.

Oxidative stress and immune dysfunction are linked to both Hashimoto's thyroiditis and polycystic ovary syndrome, with diet and specific treatments important for managing these conditions.

The conclusion is that obesity should be managed with a slow, balanced approach to diet and exercise, with medication and surgery as additional options, and education and access to care are important.

Inclusive and culturally competent healthcare is crucial for transgender and non-binary individuals.

Enterococcus faecalis delays wound healing by disrupting cell functions and creating an anti-inflammatory environment.

A woman's rare benign tumor that released both cortisol and testosterone was successfully removed, improving her health.

Cellular and immunotherapies show promise for healing chronic wounds but need more research.

Exosomal miRNAs from stem cells can help improve skin health and delay aging.

Adipose-derived stem cells can help repair and improve eye tissues and appearance.