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Nanotechnology is improving drug delivery and targeting, with promising applications in cancer treatment, gene therapy, and cosmetics, but challenges remain in ensuring precise delivery and safety.

Mongolian gerbils heal wounds differently than mice, with unique protein levels and gene expression that affect skin repair.

KDML105 bran extract may help with hair growth and prevent hair loss.

Removing Mediator 1 from certain mouse cells causes teeth to grow hair instead of enamel.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

New tools show that in fish, NPY increases feeding and somatostatin decreases it.

The conclusion is that the nuclear lamina and LINC complex in skin cells respond to mechanical signals, affecting gene expression and cell differentiation, which is important for skin health and can impact skin diseases.

Deleting the CRIF1 gene in mice disrupts skin and hair formation, certain proteins affect hair growth, a new compound may improve skin and hair health, blood cell-derived stem cells can create skin-like structures, and hair follicle stem cells come from embryonic cells needing specific signals for development.

K_ATP channel gene mutations are linked to heart diseases, but more research is needed to understand the connection and treatment potential.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Mice without the Sp6 gene have problems developing several body parts, including hair, teeth, limbs, and lungs.

Hairless protein affects hair follicle structure by regulating the Dlx3 gene.

Nanog gene boosts stem cells, helps hair growth, and may treat hair loss.

A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

The OVOL1 gene, controlled by β-catenin, is crucial for creating hair follicles.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Deleting the PTEN gene in mice causes nerve cells to grow larger and heal better after injury, but may cause overgrowth and hair loss in older mice.

The protein STAT3 slows down cell growth by blocking the FST gene, which affects hair development in sheep.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

SP1 promotes and KROX20 inhibits hair cell growth by affecting the CUX1 gene.

Tet2 and Tet3 enzymes are essential for controlling hair growth by affecting DNA demethylation and gene expression in mice.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

Researchers found a genetic link for hereditary hair loss but need more analysis to identify the exact gene.

The gene Tfap2b is essential for creating a type of stem cell in zebrafish that can become different pigment cells.

Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

The Hairless gene is crucial for healthy skin and hair growth.

Testing for CAG repeat polymorphism in the androgen receptor gene is not currently recommended for managing hypogonadism.