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Deleting Snai2 and Snai3 causes fatal autoimmunity.

ALDOA levels drop in hair cells during hair loss.

Propylthiouracil can cause hair loss, reduced sweating, and nail issues, but stopping the drug can reverse hair and nail problems.

New findings suggest potential treatments for melanoma, hyperpigmentation, hair defects, and multiple sclerosis, and show skin microbiome changes don't cause atopic dermatitis.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

Langerhans cells and melanocytes migrate to the skin and hair follicles during early human development.

Fat tissue-derived cells show promise for repairing body tissues, but more research and regulation are needed for safe use.

The skin has a complex immune system that is essential for protection and healing, requiring more research for better wound treatment.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

Some drugs can cause rare but potentially deadly skin reactions, and early treatment and avoiding the drug again are key.

The secretome from mesenchymal stem cells is a promising treatment that may repair tissue and avoid side effects of stem cell transplantation.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

Men generally have more severe COVID-19 cases and higher death rates than women due to biological differences.

Men, especially older ones with health issues like prostate cancer, may have worse COVID-19 outcomes and could benefit from therapies targeting male hormones.

Restoring hair bulb immune privilege is crucial for managing alopecia areata.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

Mice studies show that Protein Phosphatase 2A is crucial for cell growth, development, and disease prevention.

Pregnancy can increase lupus activity, but careful planning and treatment can improve outcomes.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

An imbalance between certain immune cells is linked to a chronic skin condition and may be influenced by obesity, smoking, and autoimmune issues.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

A deficiency in the TTC7A gene causes immune problems, gut issues, and hair loss.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

TGFβ's manipulation of inflammation and immune cells affects cancer spread, suggesting new treatment strategies and biomarkers.

Valproic Acid could potentially be used to treat immune-related conditions due to its ability to modify immune cell functions.

Lymphatic vessels are essential for hair follicle growth and skin regeneration.

Blocking JAK/STAT pathways can help treat hair loss from alopecia areata.