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Anti-acne medications may work by reducing the activity of a protein involved in acne development.

Increased FGFR2b signaling, influenced by androgens, plays a role in causing acne.

A gene variation is linked to hair thickness in Asians.

The new anti-acne treatment HA-P5 effectively reduces acne by targeting two key receptors and avoids an enzyme that can hinder treatment.

FGFR2 signaling controls Merkel cell formation in different skin regions.

A device was made in 2008 to measure hair loss severity. Other findings include: frizzy mutation in mice isn't related to Fgfr2, C/EBPx marks preadipocytes, Cyclosporin A speeds up hair growth in mice, blocking plasmin and metalloproteinases hinders healing, hyperbaric oxygen helps ischemic wound healing, amniotic membranes heal wounds better than polyurethane foam, rhVEGF165 from a fibrin matrix improves tissue flap viability and induces VEGF-R2 expression, and bFGF enhances wound healing and reduces scarring in rabbits.

The document concludes that certain genetic mutations and dietary factors are involved in acne development, and treatments like isotretinoin and diet changes can help manage it.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Keloid scars may form due to changes in skin cell characteristics and specific protein signaling.

Genetics play a role in acne, but how exactly they contribute is not fully understood.

Eating high glycemic foods and drinking milk may worsen acne by increasing insulin and IGF-1 levels.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

CD61 is important for mouse tooth cell growth and works through Lgr5.

A peptide from hair follicle stem cells promotes hair growth by activating specific skin cells.

Understanding how acne develops in different diseases could lead to new treatments.

Studying acne-related syndromes helps us understand acne causes and can lead to new treatments.

Genetic factors play a major role in acne.

A new protein was made to detect specific skin cell growth receptors and worked in normal skin but not in skin cancer cells.

The study found that a specific signaling pathway helps skin wounds heal faster but may lead to larger scars.

Hedgehog signaling is crucial for mammary gland development over hair follicles.

The document concludes that understanding the sebaceous gland's development and function is key to addressing related skin diseases and aging effects.

Growth factors and cytokines are important for hair growth and could potentially treat hair loss, but more research is needed to overcome challenges before they can be used in treatments.

RNA aptamers can specifically block FGF5-related cell growth, potentially treating related diseases or hair disorders.

Increasing the levels of a protein called FGF9 can promote hair growth, but humans may not respond the same way due to a lack of certain cells.

Acne is linked to inflammation and insulin resistance, and is associated with various syndromes that require different treatments.

Li2CO3 improved skin disease in a mouse model of Focal Dermal Hypoplasia without toxicity.

A rare skin disorder, nevus comedonicus, can appear on one side of the body following Blaschko's lines.

Skin symptoms can indicate endocrine disorders and have various treatments.

The document concludes that skin stem cells are important for hair growth and wound healing, and could be used in regenerative medicine.

Hair follicle development is controlled by interactions between skin tissues and specific molecular signals.