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Bone-marrow and epidermal stem cells help heal wounds differently, with bone-marrow cells aiding in blood vessel formation and epidermal cells in hair growth.

Vitamin D and calcium are important for quick and effective skin wound healing.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

Dermal EZH2 controls skin cell development and hair growth in mice.

Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

Dermal EZH2 controls skin cell growth and differentiation in mice.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

Mitochondrial reactive oxygen species are essential for skin and hair development.

Epigenetic mechanisms control skin development by regulating gene expression.

Wnt signaling is crucial for skin development and hair growth.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

Skin side effects from cancer drugs targeting EGFR can affect treatment adherence but can be managed with antibiotics like tetracycline.

ASH2L is essential for skin and hair development.

Removing PLCg1 from skin cells caused thicker oil glands and less hair in mice.

TRPV3 in skin cells causes inflammation and cell death.

Skin can heal wounds without hair follicle stem cells, but it takes a bit longer.

Claudin-1 is important for the barrier function and growth of hair.

The congress showed advancements in skin hydration, barrier function, and safe, effective new cosmetic formulations.

Certain fats in the skin help control inflammation and health, and changing these fats through diet or supplements might treat skin inflammation.

Fat-derived stem cells show promise for skin repair and reducing aging signs but need more research for consistent results.

Endo-HSE helps grow hair-like structures from human skin cells in the lab.

Different oil products cause varying levels of skin irritation in mice, which could potentially lead to tumors.

Non-thermal plasma treatment makes mouse skin thicker and increases growth factors without harming the tissue.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

COX-2 levels change during the hair cycle and affect skin and hair growth.

Liposomal systems show promise for delivering drugs through the skin but face challenges like high costs and stability issues.

TRP channels in the skin are important for sensation and health, and targeting them could help treat skin disorders.

Wnt and Notch signaling help wound healing by promoting cell growth and regulating cell differentiation.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

Cystatin M/E strongly inhibits cathepsin V and cathepsin L, important for skin formation.