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Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

ACBP is crucial for healthy skin in mice.

The protein Par3 is crucial for healthy skin, affecting the skin barrier, cell differentiation, and stem cell maintenance.

Keratosis pilaris is often linked to genetic mutations and causes skin and hair abnormalities, regardless of those mutations.

ACBP is essential for healthy skin and fur by maintaining the skin's barrier function.

PPARγ is essential for maintaining healthy skin, controlling inflammation, and ensuring proper skin barrier function.

Enzymes that change arginine to citrulline are important for skin barrier and hair formation, and their malfunction can lead to skin conditions and hair disorders.

Glucosylceramides are essential for healthy skin and proper wound healing.

Healthy skin prevents water loss and protects against threats.

Dsg1 is essential for maintaining a healthy skin barrier in mice.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

LSD1 is essential for healthy skin development and creating the skin's protective barrier.

The STRIPAK complex is crucial for skin cell organization and creating a functional skin barrier.

Dry skin does not mean the skin's protective barrier is broken; it's often due to a lack of natural skin moisturizers.

Cholesterol sulfate buildup due to a genetic mutation disrupts the skin barrier, leading to the scaling skin seen in X-linked ichthyosis.

Hataedock treatment improved skin health and reduced atopic dermatitis symptoms by enhancing the skin barrier and reducing inflammation.

Fatp4 is crucial for healthy skin development and function.

Stem cell differentiation is crucial for skin barrier maintenance and its disruption can lead to skin diseases.

A specific gene variant causes severe skin issues and increases infection risk, requiring careful medical monitoring.

The enzyme DHHC13 is essential for healthy hair and skin, and its deficiency leads to hair loss and skin problems.

The PP2A-B55α protein is essential for brain and skin development in embryos.

ILK is essential for skin development, pigmentation, and healing.

Radiation therapy damages skin structure and immune function, causing inflammation and potential hair loss.

The synthetic retinoid EC23 thickens skin and promotes hair growth more effectively and with a lower dose than natural retinoids.

EGFR inhibitors cause skin issues and hair loss by weakening skin defenses, suggesting antibiotics and targeted treatments can help.

EAAT4 decreases with age, harming skin function and calcium balance.

Key skin cell regulators and gene organization changes are crucial for skin cell development and could help treat skin disorders.

Esrp1 is important for skin health by helping form and maintain the skin barrier.

Epidermal keratinocytes start wound healing and inflammation after schistosome infection.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.