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Skin healing from blisters can delay hair growth as stem cells focus on repairing skin over developing hair.

Alopecia areata is likely caused by a combination of genetic factors and immune system dysfunction, and may represent different diseases with various causes.

Stress can worsen skin conditions by affecting hormone levels and immune response.

People with atopic dermatitis are more likely to develop other skin conditions due to shared genetics and immune pathways.

New protein changes may be involved in the immune attack on hair follicles in alopecia areata.

The skin has multiple layers and cells, serves as a protective barrier, helps regulate temperature, enables sensation, affects appearance, and is involved in vitamin D synthesis.

Ingenol mebutate gel changes gene expression related to skin development and immune response in actinic keratosis.

Using gelatin sponges for deep skin wounds helps bone marrow cells repair tissue without scarring.

Genes controlling hair growth and immune response are disrupted in male pattern baldness.

Alopecia areata causes patchy hair loss and involves immune system disruptions.

Certain mice without specific receptors or mast cells don't lose hair from stress.

Fibroblasts, cells usually linked to tissue repair, also help regenerate various organs and their ability decreases with age. Turning adult fibroblasts back to a younger state could be a new treatment approach.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Certain drugs increase calcium levels in cancer cells by triggering internal calcium release.

The research created a detailed map of skin cells, showing that certain cells in basal cell carcinoma may come from hair follicles and could help the cancer grow.

The skin is a complex barrier that protects the body, regulates temperature, and helps with immune responses.

Applying certain inhibitors to the skin can promote hair growth without harming cells.

Future research on ectodysplasin should explore its role in diseases, stem cells, and evolution, and continue developing treatments for genetic disorders like hypohidrotic ectodermal dysplasia.

Pure carbon nanotubes are safe for mice, but impure ones cause immune issues and hair loss.

Nanoparticles around 600-700 nm can effectively enter and stay in hair follicles for days, which may help in delivering drugs to specific cells.

Foxp3+ Regulatory T Cells are important for immunity and tolerance, affect hair growth and wound healing, and their dysfunction can contribute to obesity-related diseases and other health issues.

Fat tissue under the skin affects hair growth and aging; reducing its inflammation may help treat hair loss.

Four specific genes are linked to keloid formation and could be potential treatment targets.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

Certain microRNAs may protect against hair loss in alopecia areata and could be potential treatment targets.

Foxn1 is important for fat development, metabolism, and wound healing in skin.

A virus protein can activate a pathway that may lead to abnormal hair follicle development.

Fibroblast state switching is crucial for skin healing and development.

Macrophages are vital for skin healing, hair growth, salt balance, and cancer defense.

Melanocyte stem cells are crucial for skin pigmentation and have potential in disease modeling and regenerative medicine.