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Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Inherited ichthyoses cause widespread skin scaling and thickening due to gene mutations.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

Most adrenal cortex tumors are benign and non-secreting, but proper diagnosis and treatment are important due to the rare possibility of cancer.

New genotype linked to non-classical congenital adrenal hyperplasia found in Italian siblings.

A new mutation in the CYP11B1 gene was found in a woman with mild hyperandrogenemia, a rare cause of non-classic congenital adrenal hyperplasia.

Hairless protein can block vitamin D activation in skin cells.

Non-classical 21 hydroxylase deficiency is an underdiagnosed cause of female hair loss and polycystic ovarian syndrome.

The skin acts like an endocrine organ, producing hormones that affect various body functions and skin health, and understanding this can lead to new treatments.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Acne is caused by genetics, diet, hormones, and bacteria, with treatments not yet curative.

ZDHHC13 is important for normal liver function and metabolism, affecting mitochondrial activity.

Eating healthy, exercising, and taking certain supplements can help manage Polycystic Ovary Syndrome symptoms.

Certain gene variants may contribute to high androgen levels in women with polycystic ovary syndrome.

Human skin produces sex hormones like estrogen and testosterone, influenced by ARO and StAR, which may affect skin elasticity and hair growth.

PFS and PSSD are similar conditions with persistent sexual dysfunction after stopping medication.

Certain fats in the skin help control inflammation and health, and changing these fats through diet or supplements might treat skin inflammation.

The meeting focused on understanding, diagnosing, and finding treatments for irreversible hair loss diseases.

Altered retinoid metabolism in cicatricial alopecia suggests a balanced vitamin A diet may prevent the condition.

A gene in Sebekia benihana, CYP-sb21, is needed for a specific reaction on the drug Cyclosporine A, which could be important for hair growth without affecting the immune system.

Researchers found that most genes affecting drug responses are not fully covered by commercial SNP chips, suggesting the need for more comprehensive tools to optimize drug selection based on genetics.

The document concludes that managing PCOS involves treating symptoms and reducing long-term metabolic risks, with lifestyle changes being important.

Selective breeding caused the unique curly hair in Mangalitza pigs.