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People with alopecia areata have more Th17 cells and fewer Treg cells, which may be key to the condition's development.

The secretions of mesenchymal stem cells could be used for healing without using the cells themselves.

Current and future treatments for alopecia areata focus on immunosuppression, immunomodulation, and protecting hair follicles.

In vivo lineage labelling is better than in vitro methods for identifying and understanding stem cells.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

Hormones during pregnancy and lactation keep skin stem cells inactive, preventing hair growth.

Male hormones cause different growth in identical human hair follicles due to their unique epigenetic characteristics.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

Both Th1 and Th2 immune responses are increased in alopecia areata, with Th2 response more strongly linked to how severe the disease is.

Activating Nrf2 helps wounds heal faster by increasing hair follicle stem cells.

Misbehaving hair follicle stem cells can cause hair loss and offer new treatment options.

Treg dysfunction is linked to various autoimmune skin diseases, and understanding Treg properties is key for new treatments.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Different types of skin cells play specific roles in development, healing, and cancer.

TGF-β is crucial for controlling stem cell behavior and changes in its signaling can lead to diseases like cancer.

Different types of stem cells and their environments are key to skin repair and maintenance.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Muscles and nerves that cause goosebumps also help control hair growth.

Skin cell-derived vesicles can help heal skin injuries effectively.

Hair follicle stem cells are a promising source for tissue repair and treating skin or hair diseases.

Different cell types work together to repair skin, and targeting them may improve healing and reduce scarring.

Aconiti Ciliare Tuber extract may help hair grow by activating a specific cell signaling pathway.

Older hair follicle stem cells have a reduced ability to renew themselves, leading to more hair loss.

Fzd2 is important for skin and hair development through various signaling ways.

UK medical students lack dermatology education, liver biopsy patients with risk factors show more fibrosis, and certain fungi resist drugs due to melanin; genetics may influence female hair loss.

The Wnt/β-catenin pathway helps tissue regeneration but can also cause fibrosis, and drugs that inhibit this pathway may aid in healing skin and heart tissues.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Skin stem cells are crucial for maintaining and repairing the skin and hair, using a complex mix of signals to do so.

Wnt signaling helps control how brain stem cells divide and is important for brain repair after injury.

Alopecia Areata is an autoimmune disease affecting hair follicles, influenced by genetic and environmental factors, with rodent models being essential for research.