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Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

Scientists turned rat thymus cells into stem cells that can help repair skin and hair.

We need more research to better understand human hair follicle stem cells for improved treatments for hair loss and skin cancer.

Thymosin β4 promotes hair growth by activating stem cells in hair follicles.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

Blocking β-catenin in skin cells improves hair growth during wound healing.

Keratin 15 is not a reliable sole marker for identifying epidermal stem cells because it's found in various cell types.

The skin protects the body and is constantly renewed by stem cells; disruptions can lead to cancer.

The article concludes that while we understand a lot about how melanocytes age and how this can prevent cancer, there are still unanswered questions about certain pathways and genes involved.

Skin development in mammals is controlled by key proteins and signals from underlying cells, involving stem cells for maintenance and repair.

Muscles and nerves that cause goosebumps also help control hair growth.

LGR5 helps maintain corneal cell characteristics and prevents unwanted changes by controlling specific cell signaling pathways.

Shrinking skin cancer increases the chance of cancer in nearby lymph nodes.

Androgen receptors are key for development and health, affecting conditions like prostate cancer and male pattern baldness.

Key genes are crucial for Drosophila wing development and could be insecticide targets.

Scarring alopecia affects different hair follicle stem cells than nonscarring alopecia, and the infundibular region could be a new treatment target.

Wounds on the face usually heal with scars, but understanding how some wounds heal without scars could lead to better treatments.

Modified rat stem cells on a special scaffold improved blood vessel formation and wound healing in skin substitutes.

Researchers isolated a new type of stem cell from mouse skin that can renew itself and turn into multiple cell types.

PDGF signaling is crucial for maintaining and renewing hair follicle stem cells, which could help treat hair loss.

The protein SLC1A3 is important for activating skin stem cells and is necessary for normal hair and skin growth in mice.

Minoxidil affects cell growth in two ways: low doses increase growth, while high doses slow it down and can be toxic.

Transit-amplifying cells are crucial for tissue repair and can contribute to cancer when they malfunction.

Two specific hair keratin genes are active during hair growth and decline as hair transitions to rest.

Stem cells and their surrounding environment in hair follicles work closely together, affecting hair growth and having implications for cancer and tissue regeneration.

The Ovol2-Zeb1 circuit is crucial for skin healing and hair growth by guiding cell movement and growth.

Hair follicles are valuable for regenerative medicine and wound healing.

Proper control of β-catenin activity is crucial for development and preventing diseases like cancer.

Dogs could be good models for studying human hair growth and hair loss.