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Newborn skin cells can change into wound-healing cells more easily than adult ones, which might explain why baby skin heals without scars. Understanding this could help treat chronic wounds and prevent scarring.

The study found that p63 needs signals from morphogens to help skin cells differentiate properly.

Tet2 and Tet3 enzymes are important for controlling hair growth and shape by affecting gene activity and DNA structure in hair follicles.

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

Key skin cell regulators and gene organization changes are crucial for skin cell development and could help treat skin disorders.

The research provided new insights into the genetic factors contributing to hair loss and skin conditions by analyzing individual cells from the human scalp.

Tet2 and Tet3 enzymes are essential for controlling hair growth by affecting DNA demethylation and gene expression in mice.

BRG1 is essential for skin cells to move and heal wounds properly.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

Hair follicle cells change their DNA packaging during growth cycles and when grown in the lab.

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

CTCF protein is essential for skin and hair follicle development in mice.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

Nuclear shape and chromatin changes affect gene expression in skin cell differentiation.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Different small areas within hair follicles send specific signals that control what type of cells stem cells become.

The p53 protein has complex, sometimes contradictory functions, including tumor suppression and promoting cell survival.

Understanding how EDC genes are regulated can help develop better drugs for skin diseases.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.

Stem cell differentiation is crucial for skin barrier maintenance and its disruption can lead to skin diseases.

The research suggests that autophagy-related genes might play a role in causing alopecia areata.

Understanding how epigenetic regulation affects stem cells is key to cancer insights and new treatments.

A specific DNA region is crucial for Foxn1 gene expression in thymus cells but not in hair follicles.

Key genes can rewire networks, changing skin appendage types.

SOX9 helps determine stem cell roles by interacting with DNA and proteins that control gene activity.

The Hr protein binds to DNA, interacts with p53, and affects cell cycle genes.

Epigenetic changes are crucial for hair follicle stem cells to function properly.

Keratin protein production in cells is controlled by a complex system that changes with cell type, health, and conditions like injury or cancer.

The protein STAT3 slows down cell growth by blocking the FST gene, which affects hair development in sheep.