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Research 61–90 of 1000+
- High-throughput T cell receptor sequencing identifies clonally expanded CD8+ T cell populations in alopecia areata
- High throughput T cell receptor sequencing identifies clonally expanded CD8+ T cell populations in Alopecia Areata
- SOCS3 treatment prevents the development of alopecia areata by inhibiting CD8+ T cell-mediated autoimmune destruction
- Increased blood levels of NKG2D+CD4+ T cells in patients with alopecia areata
- Th1 effector CD4 T cells rely on IFN-γ production to induce alopecia areata
- Single-cell analysis of temporal immune cell dynamics in alopecia areata reveals a causal role for clonally expanded CD8+ T cells in disease
- Integrated single-cell chromatin and transcriptomic analyses of peripheral immune cells in patients with alopecia areata
- 0036 IL-27 generates immunosuppressive CD4 T cells and and prevents the development of alopecia areata
- Possible Involvement of Skin‐Resident Memory T Cells in Refractory Chronic Alopecia Areata
- 051 γδ T cells as novel players in alopecia areata pathobiology: Vδ1 + T lymphocytes may recognize “stressed” hair follicle keratinocytes, leading to IFNγ-dependent hair follicle dystrophy and immune privilege collapse
- T <sub>H</sub> 1 effector CD4 T cells rely on IFN-γ production to induce alopecia areata
- 382 Environmental pathobiology of frontal fibrosing alopecia (FFA): Does linalool promote FFA development and progression by inducing epithelial hair follicle stem cell damage and MICA expression in the bulge?
- ESDR382 - Environmental pathobiology of frontal fibrosing alopecia (FFA): Does linalool promote FFA development and progression by inducing epithelial hair follicle stem cell damage and MICA expression in the bulge?
- Alopecia areata
- Inhibition of T-cell activity in alopecia areata: recent developments and new directions
- The role of lymphocytes in the development and treatment of alopecia areata
- Performance exploration of multi-gene panels of alopecia areata susceptibility and drug-binding targets
- 455 Molecular correlation with clinical outcomes in an open label clinical trial of oral tofacitinib in patients with alopecia areata
- Dandruff lesional scalp skin exhibits epidermal T cell infiltration and a weakened hair follicle immune privilege
- Induction of T cell exhaustion by JAK1/3 inhibition in the treatment of alopecia areata
- An immune regulatory CCT repeat containing oligodeoxynucleotide capable of causing hair loss in male mice
- Treatment of Alopecia Areata in Mice by Stimulating the Hair Follicles Using Parathyroid Hormone Agonists Linked to a Collagen Binding Domain
- Involvement of ILC1-like innate lymphocytes in human autoimmunity, lessons from alopecia areata
- Hair follicle immune privilege and its collapse in alopecia areata
- Unveiling the Effect of Age and IgE Level on Alopecia Areata: Insights from Comparative RNAseq Analysis
- LB708 ILC1-like innate lymphocytes in human autoimmunity: Lessons from Alopecia Areata
- Selective inhibition of JAK3 signaling is sufficient to reverse alopecia areata
- Translational Positioning of Janus Kinase (JAK) Inhibitors in Alopecia Areata
- 315 Butyrophilin-like 2 (BTNL2) controls auto-aggressive dermal γδT lymphocytes that attack human hair follicles.
- 1406 Environmental pathobiology of frontal fibrosing alopecia (FFA): A link between linalool sensitization and FFA development